Results 121 to 130 of about 1,351 (168)
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Enteric resistance and coxsackievirus B

The American Journal of Clinical Nutrition, 1977
Sidney Kibrick,4 M.D., Ph.D. and Roger M. Loria, Ph.D.Although the oro- and nasopharynx serveas portals of entry for many different vi-nuses, the number of these agents that reachthe gut and initiate primary infection at thissite is limited. Many swallowed viruses areprobably inactivated by the acidic and pro-teolytic secretions of the stomach that ...
S, Kibrick, R M, Loria
openaire   +2 more sources

Infection of Hypercholesterolemic Mice with Coxsackievirus B

Journal of Infectious Diseases, 1976
Adult male mice were made hypercholesterolemic by a diet high in cholesterol, cholic acid, animal fat, and sucrose. After three months on this diet, animals were infected with 5 X 10(9) plaque-forming units of coxsackievirus B5. Control groups consisted of uninfected hypercholesterolemic mice and infected mice maintained on a standard laboratory diet ...
R M, Loria, S, Kibrick, G E, Madge
openaire   +2 more sources

Myelitis due to coxsackievirus B infection

Neurology, 1995
Enteroviruses, including polioviruses, coxsackieviruses A and B, echoviruses, and more recently discovered enteroviruses, are neurotropic, but there are only a few cases of flaccid paralysis reported in association with coxsackievirus. We present a 12-year-old girl with an unusual clinical presentation of this rare complication.
Jadoul, C.   +2 more
openaire   +3 more sources

Coxsackievirus B infection and pathogenesis of type 1 diabetes

Virologie, 2022
Epidemiological and experimental studies suggest that enteroviruses (EV) and particularly coxsackieviruses B (CVB) are likely to trigger or accelerate the onset of islet autoimmunity and the development of type 1 diabetes (T1D) in genetically susceptible individuals.
Nekoua, Magloire Pandoua   +10 more
openaire   +2 more sources

Group B Coxsackievirus Virulence

2008
That which is understood of virulence phenotypes in the picornaviruses derives in large part from studies of artificially attenuating phenotypes rather than through examination of naturally occurring virus strains. The CVB replicate well in a variety of different murine and human cell cultures, making them excellent viruses with which to engage the ...
S, Tracy, C, Gauntt
openaire   +2 more sources

Pathophysiological aspects of coxsackievirus B intestinal infection

The American Journal of Clinical Nutrition, 1977
Our findings reveal that intestinal infection with coxsackie B5 results in decreased intestinal epithelial cell division in association with an increase in carbohydrate (glucose) and amino acid (leucine) absorption in the small intestine. These findings are contrasted with those occurring during Salmonella infection, which results in increased ...
R M, Loria, S, Kibrick, S A, Broitman
openaire   +2 more sources

The Mechanism of Chronic Coxsackievirus B Hepatitis in Rabbits

Viral Immunology, 1999
The pathophysiology of chronic hepatitis in rabbits infected with coxsackievirus B5 (CVB5), (strain Mitchell) was investigated. Three-week-old male New Zealand White rabbits were inoculated intraperitoneally with 1 x 10(5) plaque forming units of virus.
D M, See   +4 more
openaire   +2 more sources

Treatment of viral myocarditis caused by coxsackievirus B

American Journal of Health-System Pharmacy, 2008
The treatment options for viral myocarditis caused by coxsackievirus B are summarized.Myocarditis is a common cause of dilated cardiomyopathy. The most common causes of infectious myocarditis are viruses. The exact mechanism of coxsackievirus B-induced damage to myocytes is unknown.
Luigi, Brunetti   +1 more
openaire   +2 more sources

Coxsackievirus B cardiopathy and angiopathy in the hypercholesterolemic host

Atherosclerosis, 1978
Studies on the pathogenic potential of the human cardiotropic enterovirus, coxsackievirus B5, show that this agent localizes and replicates in the aorta of mice. Nutritionally-induced hypercholesterolemia leads to an increased replication and persistence of virus in tissues, specifically the aorta.
A E, Campbell, R M, Loria, G E, Madge
openaire   +2 more sources

Coxsackievirus B: The important agent of hand, foot, and mouth disease

Journal of Medical Virology, 2023
AbstractHand, foot, and mouth disease (HFMD) is a common pediatric infectious illness caused by enteroviruses (EVs). EV‐A serotypes are the main pathogens associated with HFMD. In this study, 213 stool samples from 213 children with severe HFMD in Yunnan, China in 2013, 2015, and 2016 were further analyzed retrospectively for EV‐B infection. A total of
Yuhan Liu   +8 more
openaire   +2 more sources

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