Results 71 to 80 of about 18,979 (223)

What Counts as Educational Innovation in Clinical Anatomy?

open access: yesClinical Anatomy, EarlyView.
ABSTRACT Clinical anatomy has become a prominent setting for educational innovation because it links anatomical science with diagnostic imaging, procedural practice, surgery, simulation, and patient safety. However, the term innovation is used inconsistently.
Juan A. Sanchis‐Gimeno   +2 more
wiley   +1 more source

Loss of Twist1 leads to disruption of ciliary length, endocytic vesicle dynamics, and cell–cell junctions during neural tube formation

open access: yesDevelopmental Dynamics, EarlyView.
Abstract Background Endocytosis constitutes a fundamental cellular process governing development through coordinated regulation of plasma membrane remodeling and ciliogenesis, processes essential for cell shape changes and tissue development. Although Twist1 null embryos display complete cranial neural tube (NT) closure defects and conditional knockout
Derrick Thomas   +8 more
wiley   +1 more source

Augmented reality and physical hybrid model simulation for preoperative planning of metopic craniosynostosis surgery.

open access: yesNeurosurgical Focus, 2020
OBJECTIVE The main objective of neurosurgery is to establish safe and reliable surgical techniques. Medical technology has advanced during the 21st century, enabling the development of increasingly sophisticated tools for preoperative study that can be ...
Giselle Coelho   +11 more
semanticscholar   +1 more source

Craniosynostosis of coronal suture in Twist1+/- mice occurs through endochondral ossification recapitulating the physiological closure of posterior frontal suture

open access: yesFrontiers in Physiology, 2011
Craniosynostosis, the premature closure of cranial suture, is a pathologic condition that affects 1/2000 live births. Saethre-Chotzen syndrome is a genetic condition characterized by craniosynostosis.
Bjorn eBehr   +3 more
doaj   +1 more source

Secretopathies emerge as a new class of neurocristopathies

open access: yesDevelopmental Dynamics, EarlyView.
Abstract Neural crest cells are a transient embryonic population of cells that give rise to a wide range of structures, including craniofacial cartilage and bone, peripheral neurons and glia, as well as components of the cardiac outflow tract, among others.
Amanda Teixeira   +3 more
wiley   +1 more source

Cranial Neural Crest Cells and Their Role in the Pathogenesis of Craniofacial Anomalies and Coronal Craniosynostosis

open access: yesJournal of Developmental Biology, 2020
Craniofacial anomalies are among the most common of birth defects. The pathogenesis of craniofacial anomalies frequently involves defects in the migration, proliferation, and fate of neural crest cells destined for the craniofacial skeleton.
Erica Siismets, N. Hatch
semanticscholar   +1 more source

Mapping the Landscape of Over‐Scanning in CT Imaging: A Scoping Review

open access: yesJournal of Medical Radiation Sciences, EarlyView.
Over‐scanning in CT is highly prevalent and contributes to unnecessary radiation exposure, with notable impact on radiosensitive organs. Standardised protocols and AI‐assisted planning show strong potential to optimise scan range and reduce excess dose.
Mo'men Bani‐Ahmad   +5 more
wiley   +1 more source

Similarities and differences between IL11 and IL11RA1 knockout mice for lung fibro-inflammation, fertility and craniosynostosis

open access: yesScientific Reports, 2020
Loss of function (LOF) in IL11RA infers IL11 signaling as important for fertility, fibrosis, inflammation and incompletely penetrant craniosynostosis. The impact of LOF in IL11 has not been characterized.
B. Ng   +9 more
semanticscholar   +1 more source

Eustachian Tube Opening in Children With and Without Cleft Palate and Association With Otitis Media

open access: yesThe Laryngoscope, EarlyView.
Using transnasal endoscopy in 50 children, structural abnormalities of the torus tubarius—particularly the posterior lip—were significantly more common in children with cleft palate than in those without (52% vs. 20%). Adenoid size, dynamic Eustachian tube opening visibility, and the Eustachian Tube Inflammation Scale correlated with middle ear status ...
Kawin Atipas   +4 more
wiley   +1 more source

Sp1-activated FGFR2 is involved in early-life exposure to nickel-induced craniosynostosis by regulating the ERK1/2 signaling pathway

open access: yesEnvironment International
Nickel, a common environmental hazard, is a risk factor for craniosynostosis. However, the underlying biological mechanism remains unclear. Here, we found that early-life nickel exposure induced craniosynostosis in mice.
Zhenkun Weng   +11 more
doaj   +1 more source

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