Results 31 to 40 of about 9,307,341 (282)

Oral manifestations of Crohn's disease [PDF]

open access: yesStomatološki glasnik Srbije, 2004
Crohn's disease is a chronic granulomatous inflammatory bowel disease of unknown cause. Segments of the whole gastrointestinal tract may be involved, including the oral cavity.
Marković Ljiljana, Ljušković Bešir
doaj   +1 more source

Molecular prediction of disease risk and severity in a large Dutch Crohn's disease cohort [PDF]

open access: yes, 2009
Item does not contain fulltextBACKGROUND: Crohn's disease and ulcerative colitis have a complex genetic background. We assessed the risk for both the development and severity of the disease by combining information from genetic variants associated with ...
Bodegraven, A.A. van   +15 more
core   +3 more sources

Autophagy and Crohn's Disease [PDF]

open access: yesJournal of Innate Immunity, 2013
Advances in genetics have shed light on the molecular basis of Crohn's disease (CD) predisposition and pathogenesis, via linkage disequilibrium analysis to genome-wide association studies. The discovery of genetic variants of <i>NOD2</i>, an intracellular pathogen molecular sensor, as risk factors for CD has paved the way for further ...
Hang Thi Thu Nguyen, -   +3 more
openaire   +5 more sources

Multicentric Cutaneous Crohn's Disease: A Case Report and Review of Literature

open access: yesCanadian Journal of Gastroenterology, 1990
A patient presented with skin lesions at sites not contiguous with Crohn's disease of the bowel and with specific histological features of Crohn's disease occurring two weeks from the onset of bowel symptoms.
Robert Prokopetz   +3 more
doaj   +1 more source

Systems biology in inflammatory bowel diseases [PDF]

open access: yes, 2014
Purpose of review: Ulcerative colitis (UC) and Crohn’s Disease (CD) are the two predominant types of inflammatory bowel disease (IBD), affecting over 1.4 million individuals in the US.
Iliopoulos, D, Koukos, G, Polytarchou, C
core   +1 more source

Crohn's disease: Th1, Th17 or both? The change of a paradigm: new immunological and genetic insights implicate Th17 cells in the pathogenesis of Crohn's disease [PDF]

open access: yes, 2009
Traditionally, Crohn's disease has been associated with a Th1 cytokine profile, while Th2 cytokines are modulators of ulcerative colitis. This concept has been challenged by the description of tolerising regulatory T cells (Treg) and by proinflammatory ...
Brand, Stephan
core   +1 more source

Metastatic Crohn’s disease despite infliximab therapy [PDF]

open access: yesAnais Brasileiros de Dermatologia, 2017
Metastatic Crohn’s disease is a rare extraintestinal manifestation of Crohn’s disease. It is characterized by polymorphic skin lesions formed by non-caseating granulomas located on anatomical sites distant from the gastrointestinal tract.
Sara Campos   +3 more
doaj   +1 more source

OC-163 identification of inflammatory bowel disease (IBD) using field asymmetric ion mobility spectrometry (FAIMS) [PDF]

open access: yes, 2012
Introduction Resident colonic bacteria, principally anaerobes and firmicutes, ferment undigested fibre. The resultant volatile organic compounds (VOCs) formed are dissolved in the faeces but also absorbed and excreted in the urine.
Arasaradnam, Ramesh P.   +7 more
core   +1 more source

The role of the novel Th17 cytokine IL-26 in intestinal inflammation [PDF]

open access: yes, 2008
Background and aims: Interleukin 26 (IL-26), a novel IL-10-like cytokine without a murine homologue, is expressed in T helper 1 (Th1) and Th17 cells. Currently, its function in human disease is completely unknown. The aim of this study was to analyse its
Auernhammer, Christoph J.   +7 more
core   +1 more source

Progress and Clinical Applications of Crohn’s Disease Exclusion Diet in Crohn’s Disease

open access: yesGut and Liver
Crohn’s disease is a chronic intestinal inflammatory disorder of unknown etiology. Although the pharmacotherapies for Crohn’s disease are constantly updating, nutritional support and adjuvant therapies have recently gained more attention.
Duo Xu   +5 more
doaj   +1 more source

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