Results 81 to 90 of about 177,396,190 (300)

Other title: Availability and utilization of health care services at acute care hospitals and federally qualified health centers fiscal year 2010

open access: yes, 2011
"As required by Connecticut General Statutes (C.G.S.) section 19a-634 this report is one of three components of the Department of Public (DPH) Health Office of Health Care Access' (OHCA) approach to planning for Connecticut's health care system"--P. 4.; "

core  

Somatostatin receptor 4 (SSTR4) is a tumor suppressor in cutaneous and head & neck squamous cell carcinomas

open access: yesMolecular Oncology, EarlyView.
This study identifies somatostatin receptor 4 (Sstr4) as a critical tumor suppressor against skin and head/neck cancers (HNSCC, cSCC, and BCC). The loss of Sstr4 removes a check on cell growth, causing hyperactivation of the MAPK‐ERK signaling pathway (↑).
Ali Taqvi   +6 more
wiley   +1 more source

Understanding medical trainees’ anti-fat dislike: roles of observed weight stigma, body image disturbance, and controllability beliefs

open access: yesBMC Psychology
Background Studies have demonstrated that medical students’ anti-fat prejudice increases over the course of training, although the mechanisms through which these prejudicial attitudes are heightened are not well understood.
Samantha R. Philip   +5 more
doaj   +1 more source

Castration‐resistant prostate cancer cells are addicted to the high activity of cyclin‐dependent kinase 2

open access: yesMolecular Oncology, EarlyView.
We show that emergence of castration‐resistant prostate (CRPC) is associated with significant upregulation of cyclins that positively regulate cyclin‐dependent kinase 2 (CDK2) and concomitant downregulation of CDK4 cyclins. This renders CRPC cells dependent on the high activity of CDK2, and CDK2 inhibitors synergistically sensitize CRPC cells to both ...
Joyeeta Chatterjee   +3 more
wiley   +1 more source

The Connecticut Office of Health Care Access 2004 Small Employer Health Insurance Survey-focus on results /

open access: yes, 2004
Title from caption.; "Planning tomorrow's health care system today."; "November 2004."; Updates: Employer-sponsored health insurance / Office of Health Care Access. Hartford, CT : Office of Health Care Access, [2001]

core   +1 more source

State Variation in Smart Device Eligibility for Aging-in-Place Services: Recent Trends in Medicaid Section 1915(c) Waiver Coverage for Older Adults

open access: yesClinical Interventions in Aging
Stephanie Zawada,1 Michelle Fogus,2 Emily Mills,3 Emma Fortune,4 Laura Cepoi21Center for Clinical and Translational Science, Mayo Clinic, Rochester, MN, USA; 2Olympic Area Agency on Aging, Aberdeen, WA, USA; 3Intuition Robotics, Seattle, WA, USA; 4Kern ...
Zawada S   +4 more
doaj  

Gut microbiota alterations in patients with non‐small‐cell lung cancer undergoing chemoradiotherapy

open access: yesMolecular Oncology, EarlyView.
We investigated the impact of concurrent chemoradiotherapy (CRT) on the gut microbiota in patients with locally advanced non‐small‐cell lung cancer. Overall gut microbiota composition remained largely stable throughout CRT while antibiotic exposure may influence microbial diversity.
Hanne Marte Nymoen   +19 more
wiley   +1 more source

Delivery of Health Care by Spanish Dental Hygienists in Private and Public Dental Services during the COVID-19 De-Escalation Phase (June 2020): A Cross-Sectional Study. [PDF]

open access: yesInt J Environ Res Public Health, 2021
Expósito-Delgado AJ   +9 more
europepmc   +1 more source

Regulation of the lncRNA NEAT1 by p53‐ΔNp63 crosstalk modulates the DNA damage response and therapeutic efficacy in HNSCC

open access: yesMolecular Oncology, EarlyView.
In head and neck squamous cell carcinoma (HNSCC) p53 and p63 exert opposite roles on the transcription regulation of the lncRNA NEAT1. Under basal conditions, p53 levels are low and p63 represses NEAT1 expression. Upon genotoxic stress, p53 is rapidly induced, displacing p63 from the NEAT1 promoter leading to NEAT1 transcriptional activation and ...
Sara De Domenico   +5 more
wiley   +1 more source

Reprogramming tumor cells with LCOR mRNA nanotherapy to enhance immunotherapy

open access: yesMolecular Oncology, EarlyView.
LCOR (Ligand‐dependent corepressor) mRNA encapsulated in polymeric nanoparticles restores LCOR activity in tumor cells, constraining aggressive tumor cell states and increasing tumor immunogenicity. This reprogramming makes tumor cells more vulnerable to T‐cell attack and enhances their response to immune‐checkpoint immunotherapy.
Gabriel Serra‐Mir   +9 more
wiley   +1 more source

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