Results 261 to 270 of about 835,318 (379)

Paired Deep Brain Stimuli Elicit Short‐Term Facilitation in Globus Pallidus Interna and Subthalamic Nucleus

Movement Disorders, EarlyView.
Abstract Background Deep brain stimulation (DBS) elicits oscillatory local field potentials in patients with Parkinson's disease (PD) and other movement disorders. Greater knowledge about the fast dynamics of these neural responses could shed light on circuit pathophysiology and inform novel approaches to neuromodulation therapies.
Sarah Brinkerhoff, Arie Nakhmani, Alex Varghese, Cameron Gordon, Christopher L. Gonzalez, J. Nicole Bentley, Marshall T. Holland, Harrison C. Walker   +7 more
wiley   +1 more source

Auditory fear memory retrieval requires BLA-LS and LS-VMH circuitries via GABAergic and dopaminergic neurons. [PDF]

EMBO Rep
Chen M, Li J, Shan W, Yang J, Zuo Z.
europepmc   +1 more source

Local circuit neurons in the striatum regulate neural and behavioral responses to dopaminergic stimulation [PDF]

, 2002
Esen Saka, Michael J. Iadarola, David Fitzgerald, Ann M. Graybiel   +3 more
openalex   +1 more source

Long‐Read Sequencing: The Third Generation of Diagnostic Testing for Dystonia

Movement Disorders, EarlyView.
Abstract Long‐read sequencing methodologies provide powerful capacity to identify all types of genomic variations in a single test. Long‐read platforms such as Oxford Nanopore and PacBio have the potential to revolutionize molecular diagnostics by reaching unparalleled accuracies in genetic discovery and long‐range phasing.
Thomas Wirth, Kishore R. Kumar, Michael Zech   +2 more
wiley   +1 more source

Dopaminergic Neurons in Zona Incerta Drives Appetitive Self-Grooming. [PDF]

Adv Sci (Weinh)
Jiang Z, He M, Young C, Cai J, Xu Y, Jiang Y, Li H, Yang M, Tong Q.   +8 more
europepmc   +1 more source

Expanding the Allelic and Clinical Heterogeneity of Movement Disorders Linked to Defects of Mitochondrial Adenosine Triphosphate Synthase

Movement Disorders, EarlyView.
Abstract Background Defects of mitochondrial ATP synthase (ATPase) represent an emerging, yet incompletely understood group of neurodevelopmental diseases with abnormal movements. Objective The aim of this study was to redefine the phenotypic and mutational spectrum of movement disorders linked to the ATPase subunit‐encoding genes ATP5F1A and ATP5F1B ...
Philip Harrer, Magdalena Krygier, Martin Krenn, Volker Kittke, Martin Danis, Georgi Krastev, Alice Saparov, Virginie Pichon, Marlène Malbos, Clarisse Scherer, Ivana Dzinovic, Matej Skorvanek, Robert Kopajtich, Holger Prokisch, Sara Silvaieh, Anna Grisold, Maria Mazurkiewicz‐Bełdzińska, Jean‐Madeleine de Sainte Agathe, Juliane Winkelmann, Jan Necpal, Robert Jech, Michael Zech   +21 more
wiley   +1 more source

Reduced Neurite Arborization in Primary Dopaminergic Neurons in Autism-Like Shank3B-Deficient Mice. [PDF]

Mol Neurobiol
Bacova Z, Havranek T, Mihalj D, Borbelyova V, Kostrubanicova K, Kramarova M, Ostatnikova D, Bakos J.   +7 more
europepmc   +1 more source

The Heart of the Matter: Cardiac Denervation Casts Doubt on the Brain‐First Versus Body‐First Hypothesis of Parkinson's Disease

Movement Disorders, EarlyView.
Tomoko Totsune, Toru Baba, Takafumi Hasegawa, Atsushi Takeda   +3 more
wiley   +1 more source

Molecular‐Informed Network Analysis Unveils Fatigue‐Related Functional Connectivity in Parkinson's Disease

Movement Disorders, EarlyView.
Abstract Background Fatigue in Parkinson's disease (PD) is a prevalent and debilitating non‐motor symptom. Despite its significant impact on quality of life, the underlying neurochemical and network‐based mechanisms remain poorly understood. Objectives This observational study applied a multimodal imaging approach to explore potential links between the
Ilaria Antonella Di Vico, Manuela Moretto, Agnese Tamanti, Giovanni Tomelleri, Giulia Burati, Daniel Martins, Ottavia Dipasquale, Mattia Veronese, Alessandra Bertoldo, Elisa Menini, Angela Sandri, Sarah Ottaviani, Francesca Benedetta Pizzini, Michele Tinazzi, Marco Castellaro   +14 more
wiley   +1 more source

Withdrawal: Neutralization of RANTES and eotaxin prevents the loss of dopaminergic neurons in a mouse model of Parkinson disease. [PDF]

J Biol Chem
Chandra G, Rangasamy SB, Roy A, Kordower JH, Pahan K.   +4 more
europepmc   +1 more source