Results 141 to 150 of about 18,651,759 (343)

Association of p120, a tyrosine kinase substrate, with E-cadherin/catenin complexes. [PDF]

open access: bronze, 1995
Sayumi Shibamoto   +9 more
openalex   +1 more source

Immunohistochemical evaluation of E-cadherin in oral epithelial dysplasia and squamous cell carcinoma

open access: gold, 2022
Jaishree Sharma   +5 more
openalex   +1 more source

Endometrial Assembloid Model Reveals Endometrial Gland Development Regulation by Estradiol‐Driven WNT7B Suppression

open access: yesAdvanced Science, EarlyView.
This study developed a 3D endometrial assembloid model to study how uterine glands form and develop. They discovered key interactions between different cell types and identified WNT7B as a regulator controlled by estradiol‐mediated TGFβ1‐VDR interaction.
Xintong Li   +12 more
wiley   +1 more source

Cleavage of E-Cadherin Contributes to Defective Barrier Function in Neosquamous Epithelium

open access: green, 2016
Thomas M. Runge   +4 more
openalex   +1 more source

A mechanically active heterotypic E-cadherin/N-cadherin adhesion enables fibroblasts to drive cancer cell invasion

open access: yesNature Cell Biology, 2017
Anna Labernadie   +13 more
semanticscholar   +1 more source

Decoding Human Placental Cellular and Molecular Responses to Obesity and Fetal Growth

open access: yesAdvanced Science, EarlyView.
Women with obesity often deliver large‐for‐gestational‐age (LGA) infants. Single‐nucleus RNA sequencing of term placenta reveals that hypoxia and TNF‐α signaling in syncytiotrophoblasts are featured in maternal obesity, but inflammatory signatures in Hofbauer cells and response to lipid or carbohydrate metabolism in fibroblasts are specific to LGA.
Hong Jiang   +12 more
wiley   +1 more source

Loss of E-cadherin promotes metastasis via multiple downstream transcriptional pathways.

open access: yesCancer Research, 2008
T. Onder   +5 more
semanticscholar   +1 more source

Human Atlas of Tooth Decay Progression: Identification of Cellular Mechanisms Driving the Switch from Dental Pulp Repair Toward Irreversible Pulpitis

open access: yesAdvanced Science, EarlyView.
Tooth decay progression transforms the dental pulp response from repair to fibrosis. At early stages, stromal cells reprogram to repair the extra cellular matrix (ECM), blood vessels, and nerves, remodel and grow, keeping repair possible. In advanced decay, hypoxia, and vessel regression, in complement with an immune switch, fuel nerve degeneration and
Hoang Thai Ha   +12 more
wiley   +1 more source

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