Results 141 to 150 of about 25,379 (184)
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Endotoxemia and neutrophil activation in vivo
American Journal of Physiology-Heart and Circulatory Physiology, 1988There is a growing body of data to suggest that marginated granulocytes mediate much of the pulmonary damage observed during endotoxemia. The mechanism(s) by which endotoxemia initiates neutrophil margination and cytotoxicity remain either controversial or unknown.
M B, Grisham, J, Everse, H F, Janssen
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Tissue oxygenation and perfusion in endotoxemia
American Journal of Physiology-Heart and Circulatory Physiology, 1996Sepsis is believed to induce disturbances in microcirculatory flow and nutrient exchange, which may result in impaired tissue oxygenation. With the use of an established rat model of endotoxemia, voltametric measurements were made of skeletal muscle (tissue) oxygen tension (PtO2) and its response to inspired oxygen concentration (FIO2).
M, Sair +4 more
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1987
The fibrinolytic process includes plasminogen cleavage by different types of plasminogen activators leading to plasmin formation. Plasmin degrades fibrin but also cartilage proteoglycan1 and activates latent collagenase2. Besides its role in hemostasis, fibrinolysis may play a role in tissue remodeling and metastasis of tumor cells3,4. The fibrinolytic
A.-M. Dosne, F. Dubor, L. Chedid
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The fibrinolytic process includes plasminogen cleavage by different types of plasminogen activators leading to plasmin formation. Plasmin degrades fibrin but also cartilage proteoglycan1 and activates latent collagenase2. Besides its role in hemostasis, fibrinolysis may play a role in tissue remodeling and metastasis of tumor cells3,4. The fibrinolytic
A.-M. Dosne, F. Dubor, L. Chedid
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Endotoxemia and endotoxemia diagnostics
Plastic and Reconstructive Surgery, 1980Koničková Z +3 more
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Surgery, gynecology & obstetrics, 1988
Endotoxemia has been incriminated as a major cause of morbidity and mortality in patients with obstructive jaundice. It has been postulated that absence of gastrointestinal bile salt flow in cholestasis enhances portal absorption of bacterial endotoxin from the intestine, thereby predisposing the host to endotoxemia and its complications. This study re-
P T, Roughneen +3 more
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Endotoxemia has been incriminated as a major cause of morbidity and mortality in patients with obstructive jaundice. It has been postulated that absence of gastrointestinal bile salt flow in cholestasis enhances portal absorption of bacterial endotoxin from the intestine, thereby predisposing the host to endotoxemia and its complications. This study re-
P T, Roughneen +3 more
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Endotoxemia and Oxidative Stressa
Annals of the New York Academy of Sciences, 1996U, Pattanaik, K, Prasad
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