Results 161 to 170 of about 23,997 (201)

The use of Biochip immunofluorescence microscopy for the serological diagnosis of epidermolysis bullosa acquisita

open access: yesArchives of Dermatological Research, 2016
Epidermolysis bullosa acquisita is a rare autoimmune bullous disease characterized by the presence of circulating antibodies directed against the collagen type VII.
Mauro Alaibac   +2 more
exaly   +2 more sources

Epidermolysis Bullosa Acquisita Responsive to Dapsone Therapy

open access: yesJournal of Cutaneous Medicine and Surgery, 2001
Background: Epidermolysis bullosa acquisita (EBA) is a chronic subepidermal blistering disease that is frequently resistant to therapy. Objective: A 58-year-old man who had a one-year history of a bullous eruption involving the hands, forearms, trunk ...
J P Callen
exaly   +2 more sources
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Epidermolysis bullosa acquisita: a case series of three paediatric patients

Clincal and Experimental Dermatology, 2022
Epidermolysis bullosa acquisita (EBA) is an acquired, subepidermal blistering skin disease characterized by autoantibodies against type VII collagen, which links the epidermis to the dermis at the dermo-epidermal junction.
Maria Laura Bageta   +11 more
semanticscholar   +1 more source

Epidermolysis bullosa acquisita with concomitant autoantibodies to α3 subunit of laminin 332

Journal of dermatology (Print), 2022
Epidermolysis bullosa acquisita (EBA) is a relatively intractable subepidermal autoimmune bullous skin disease (AIBD) caused by autoantibodies against type VII collagen.1 Antilaminin 332 (LM332)– type mucous membrane pemphigoid (MMP) is another ...
Mai Hamaguchi   +7 more
semanticscholar   +1 more source

Pediatric epidermolysis bullosa acquisita: A review

Pediatric dermatology, 2021
Epidermolysis bullosa acquisita (EBA) is an acquired autoimmune blistering skin disorder that is rare in adults and even rarer in childhood. This review aims to identify cases of pediatric EBA and report their clinical features and course. Our literature
E. Hignett, N. Sami
semanticscholar   +1 more source

Epidermolysis bullosa acquisita

Der Hautarzt, 2019
Epidermolysis bullosa acquisita (EBA) is a rare acquired subepidermal bullous autoimmune dermatosis, associated with autoantibodies against collagen type VII, the most important component of dermal anchoring fibrils. Blister induction occurs after binding of autoantibodies to collagen type VII, leading to complement activation, recruitment of ...
S C, Hofmann, A, Weidinger
openaire   +2 more sources

Evidence for a role of extracellular heat shock protein 70 in epidermolysis bullosa acquisita

Experimental Dermatology, 2021
Heat shock protein 90 (Hsp90) and Hsp70 are chaperones implicated in different inflammatory disorders, given their property to impact innate and adaptive immune responses.
S. Tukaj   +7 more
semanticscholar   +1 more source

Epidermolysis bullosa acquisita in childhood

Australasian Journal of Dermatology, 1998
SUMMARYThis case report of an 11‐year‐old girl describes a juvenile form of epidermolysis bullosa acquisita, an autoimmune disease of IgG antibodies to basement membrane type 7 collagen. Our case illustrates an unusually severe, acute inflammatory presentation of this condition with prominent mucosal and constitutional features requiring admission to a
J C, Su, G A, Varigos, J, Dowling
openaire   +2 more sources

Rituximab in the Treatment of Epidermolysis Bullosa Acquisita: A Systematic Review of the Literature.

Journal of Drugs in Dermatology
Epidermolysis bullosa acquisita (EBA) is a rare autoimmune disorder characterized by blistering of the skin and mucous membranes. Current pathophysiology implicates autoantibodies targeting type VII collagen, which serves as a crucial component of ...
Dimitra Xenopoulou   +2 more
semanticscholar   +1 more source

Pathogenesis of Epidermolysis Bullosa Acquisita

Dermatologic Clinics, 2011
Epidermolysis bullosa acquisita (EBA) is an autoimmune blistering skin disease characterized by autoantibodies to type VII collagen. Clinically, a noninflammatory and an inflammatory variant of EBA can be distinguished. Despite major achievements in the understanding of EBA, current therapeutic options are far from optimal.
Ralf J, Ludwig, Detlef, Zillikens
openaire   +2 more sources

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