Results 41 to 50 of about 23,997 (201)

Complexities of care in a pediatric patient with epidermolysis bullosa acquisita and duchenne muscular dystrophy: a case report [PDF]

open access: yesJournal of Medical Case Reports
Background Type VII collagen autoantibodies drive epidermolysis bullosa acquisita (EBA), a rare autoimmune blistering disorder. While typically seen in adults, pediatric cases—especially those with additional comorbidities—present unique clinical ...
Khayry Al-Shami   +7 more
doaj   +2 more sources

Epidermolysis bullosa acquisita as an adverse effect from rituximab therapy: A case report. [PDF]

open access: yesMedicine (Baltimore), 2020
Rationale: Rituximab is a monoclonal antibody directed against B cells and is a first-line agent for the treatment of B cell lymphoma and a second-line agent for the treatment of idiopathic thrombocytopenic purpura (ITP).
Wu X, Lv Z, Li W, Meng Z, Wan SP.
europepmc   +2 more sources

Fcγ Receptor IIB Controls Skin Inflammation in an Active Model of Epidermolysis Bullosa Acquisita. [PDF]

open access: yesFront Immunol, 2019
Epidermolysis bullosa acquisita (EBA) is an autoimmune skin blistering disease characterized by IgG autoantibodies (aAb) against type VII collagen (COL7).
Kovacs B   +8 more
europepmc   +2 more sources

Epidermolysis bullosa acquisita: current diagnosis and therapy [PDF]

open access: yesDermatology Reports, 2011
Epidermolysis bullosa acquisita (EBA) is an acquired, autoimmune subepidermal blistering disease with an approximate prevalence of 0,2/million people. The hallmark of EBA is the presence of autoantibodies (mainly IgG class) to anchoring fibril collagen ...
Christine R. Mehren, Robert Gniadecki
doaj   +2 more sources

Meta-analysis of the clinical and immunopathological characteristics and treatment outcomes in epidermolysis bullosa acquisita patients. [PDF]

open access: yesOrphanet J Rare Dis, 2018
BackgroundEpidermolysis bullosa acquisita (EBA) is an orphan autoimmune disease. Several clinical phenotypes have been described, but subepidermal blistering is characteristic of all variants.
Iwata H   +8 more
europepmc   +2 more sources

Porphyria Cutanea Tarda Masquerading as Epidermolysis Bullosa Acquisita: A Report of Two Cases

open access: yesCase Reports in Dermatology, 2015
Porphyria cutanea tarda (PCT) is the most common type of porphyria worldwide and is often initially diagnosed when cutaneous manifestations arise.
Kristyn Deen, Jason Wu
doaj   +2 more sources

Epidermolysis Bullosa Acquisita

open access: yesIndian Journal of Dermatology, 1997
A 35 years old man presented with features of epidermolysis bullosa acquisita. Here is the case report.
Gangopadhyay Asok Kumar
doaj   +3 more sources

Clinical periodontal diagnosis

open access: yesPeriodontology 2000, EarlyView., 2023
Abstract Periodontal diseases include pathological conditions elicited by the presence of bacterial biofilms leading to a host response. In the diagnostic process, clinical signs such as bleeding on probing, development of periodontal pockets and gingival recessions, furcation involvement and presence of radiographic bone loss should be assessed prior ...
Giovanni E. Salvi   +5 more
wiley   +1 more source

Diagnostic Value and Practicability of Serration Pattern Analysis by Direct Immunofluorescence Microscopy in Pemphigoid Diseases

open access: yesActa Dermato-Venereologica, 2021
In pemphigoid diseases, direct immunofluorescence can be used to differentiate 2 patterns of antibody deposition at the dermal–epidermal junction; u- and n-serrated pattern.
Maike M. Holtsche   +5 more
doaj   +1 more source

Neutrophil-specific Syk expression is crucial for skin disease in experimental epidermolysis bullosa acquisita.

open access: yesJournal of Investigative Dermatology, 2023
Autoantibodies against the dermal-epidermal junction component type VII collagen (C7) trigger skin disease in the inflammatory form of epidermolysis bullosa acquisita. We have previously identified the Syk tyrosine kinase as a crucial participant of anti-
T. Németh   +4 more
semanticscholar   +1 more source

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