Results 141 to 150 of about 22,551 (179)

Autoantibody repertoire analysis in paraneoplastic pemphigus reveals novel targets linked to mucocutaneous blistering and bronchiolitis obliterans. [PDF]

Commun Med (Lond)
Eriksson D, Aranda-Guillén M, Ishii N, Cederholm A, Behere A, Ahmed F, Katto J, Öster S, Kaipe H, Sarhan D, Kämpe O, Hashimoto T, Landegren N.   +12 more
europepmc   +1 more source

Some of the next articles are maybe not open access.

Related searches:

Epidermolysis Bullosa Acquisita (EBA) With Nonclassical Distribution of Eruptions

Journal of Dermatology, 1993
AbstractThe skin lesions in epidermolysis bullosa acquisita (EBA), a mechanobullous disease, often show acral distribution. Recently, we experienced a case of EBA in which most of the skin lesions were located on the trunk. We reviewed the distribution of the skin eruptions in 58 reported cases of EBA.
Toshihiro Tanaka, Yuji Horiguchi, S Imamura   +2 more
exaly   +3 more sources

Remission of Recalcitrant Epidermolysis Bullosa Acquisita (EBA) With Colchicine Monotherapy

Journal of Dermatology, 2005
A 42-year-old woman with widespread recalcitrant epidermolysis bullosa acquisita (EBA) responded well to colchicine monotherapy after attempts to control her disease with other modalities failed. These included dapsone, tetracycline, prednisolone, and combination of cyclophosphamide/azathioprine with dexamethasone 'pulse therapy'.
Sambit Nath Bhattacharya
exaly   +3 more sources

Comparison of 3 Type VII Collagen (C7) Assays for Serologic Diagnosis of Epidermolysis Bullosa Acquisita (EBA)

Journal of the American Academy of Dermatology, 2016
Serologic diagnosis of epidermolysis bullosa acquisita (EBA) relies on the detection of circulating autoantibodies to type VII collagen (C7).We sought to compare the diagnostic performances of a commercialized enzyme-linked immunosorbent assay (ELISA) using C7 noncollagenous (NC) domains (C7-NC1/NC2 ELISA) and indirect immunofluorescence (IIF) biochip ...
Matthias Titeux, Catherine Prost-Squarcioni, Marina Alexandre   +2 more
exaly   +3 more sources

Administration of IgG Fraction of Epidermolysis Bullosa Acquisita (EBA) Serum into Mice

The Journal of Dermatology, 1988
AbstractIn order to study the pathogenic role of autoantibodies in the serum of EBA patients, we tried to induce EBA lesions in mice by administration of the IgG fraction of EBA serum via the intraperitoneal (i.p.) or subcutaneous (s.c.) routes into neonatal or adult mice.
T, Shigemoto, K, Nashiro, T, Tsuchida, Y, Seki, K, Tamaki   +4 more
openaire   +2 more sources

Suppression of experimental epidermolysis bullosa acquisita (EBA) by an IgG monoclonal antibody (mAb) to collagen type VII (COL7)

The Journal of Immunology, 2020
Abstract EBA is a chronic inflammatory, mucocutaneous blistering skin diseases caused by autoantibodies against the structural skin protein, COL7. EBA induction requires autoantibody binding as well as complement- and Fcγ receptor (FcγR)-dependent inflammation.
Unni Krishna Srl Samavedam, Crystal Potter, Elizabeth Angerman, Anika Kasprick, Katja Bieber, Ralf Ludwig, Fred D Finkelman   +6 more
openaire   +1 more source

Manifestation der Epidermolysis bullosa acquisita (EBA) im HNO-Gebiet

HNO, 1999
Epidermolysis bullosa acquisita (EBA) is a rare, chronic, acquired bullous autoimmune dermatosis. It is characterized by the formation of IgG autoantibodies against type VII procollagen of anchoring fibrils with subepidermal formation of bullous lesions and consequent scarring.
M. Bloching, E. Dippel, S. Jovanovic, M. Hess, C. C. Zouboulis   +4 more
openaire   +1 more source

Epidermolysis bullosa acquisita (EBA)

1981
Die EBA-Diagnose stutzt sich auf folgende Kriterien: Das klinische Bild ist dem der Epidermolysis bullosa hereditaria dystrophica recessiva ahnlich. Der hauptsachliche Unterschied ist das Auftreten im reifen Alter sowie eine negative Familienanamnese.
T. Chorzelski, L. Petkow, J. Dąbrowski, T. Krainska, J. Sulej, S. Jablonska, E. H. Beutner   +6 more
openaire   +1 more source