Results 171 to 180 of about 10,384 (209)

Bushen Huoxue recipe regulates endometrial epithelial ion channels to promote embryo implantation in controlled ovarian hyperstimulation mice. [PDF]

J Chin Med Assoc
Chen ZZ, Liang W, Lu Y, Pei ST, Gou FY, Gong X.   +5 more
europepmc   +1 more source

ENAC blockade reduces blood pressure and arterial stiffness in adults with obesity and insulin resistance. [PDF]

Am J Physiol Heart Circ Physiol
Padilla J, Ramirez-Perez FI, Smith JA, Soares RN, Burken M, Uptergrove E, Lee S, Martinez-Lemus LA, Manrique-Acevedo C.   +8 more
europepmc   +1 more source

RETRACTION: Qidonghuoxue Decoction Ameliorates Pulmonary Edema in Acute Lung Injury Mice through the Upregulation of Epithelial Sodium Channel and Aquaporin-1. [PDF]

Evid Based Complement Alternat Med
And Alternative Medicine EC.
europepmc   +1 more source

The Effect of Renal Ischemia/Reperfusion on the Renal Expression of Epithelial Sodium Channels in Rat: Possible Role of Neural Precursor Cell-Expressed Developmentally Down-Regulated Protein (Nedd4-2).

Kidney Blood Press Res
Malatiali S, Fahel R, Ali RH, Redzic Z.
europepmc   +1 more source

The heterotetrameric architecture of the epithelial sodium channel (ENaC) [PDF]

EMBO Journal, 1998
The epithelial sodium channel (ENaC) is a key element for the maintenance of sodium balance and the regulation of blood pressure. Three homologous ENaC subunits (alpha, beta and gamma) assemble to form a highly Na+-selective channel. However, the subunit stoichiometry of ENaC has not yet been solved.
Ivan Gautschi, Bernard C Rossier, Laurent Schild   +2 more
exaly   +5 more sources

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Activation of the Epithelial Sodium Channel (ENaC) by Serine Proteases

Annual Review of Physiology, 2009
The study of human monogenic diseases [pseudohypoaldosteronism type 1 (PHA-1) and Liddle's syndrome] as well as mouse models mimicking the salt-losing syndrome (PHA-1) or salt-sensitive hypertension (Liddle's syndrome) have established the epithelial sodium channel ENaC as a limiting factor in vivo in the control of ionic composition of the ...
Bernard C, Rossier, M Jackson, Stutts
semanticscholar   +5 more sources

Epithelial sodium channel (ENaC) and the control of blood pressure

Current Opinion in Pharmacology, 2014
The amiloride-sensitive epithelial sodium channel (ENaC) constitutes the rate-limiting step for sodium reabsorption in epithelial cells that line the distal part of the renal tubule, the distal colon, the duct of several exocrine glands, and the lung. The activity of this channel is regulated by aldosterone and hormones involved in the maintenance of ...
B. Rossier
semanticscholar   +5 more sources

Atomic Force Microscopy Reveals the Architecture of the Epithelial Sodium Channel (ENaC) [PDF]

Journal of Biological Chemistry, 2011
The epithelial sodium channel (ENaC) is a member of the ENaC/degenerin superfamily. ENaC is a heteromultimer containing three homologous subunits (α, β, and γ); however, the subunit stoichiometry is still controversial. Here, we addressed this issue using atomic force microscopy imaging of complexes between isolated ENaC and antibodies/Fab fragments ...
Andrew P Stewart, Silke Haerteis, Alexei Diakov   +2 more
exaly   +4 more sources

Proteolytic activation of the epithelial sodium channel (ENaC) by the cysteine protease cathepsin-S [PDF]

Pflugers Archiv European Journal of Physiology, 2012
Proteolytic processing of the amiloride-sensitive epithelial sodium channel (ENaC) by serine proteases is known to be important for channel activation.
Silke Haerteis, Marko Bertog, Vera Baraznenok   +2 more
exaly   +2 more sources