Results 61 to 70 of about 495,043 (357)

Türk Kızılay’ı Bölge Kan Merkezlerinde Kan Ürünleri İmha Oranı ve Nedenleri Üzerine Tanımlayıcı Retrospektif Bir Çalışma

open access: yesAnkara Sağlık Bilimleri Dergisi
Amaç: Kan, insan hayatı için vazgeçilmez ve değerli sıvılardan biridir. Bu çalışmanın amacı Türk Kızılay’ı Kan Hizmetleri Genel Müdürlüğüne bağlı 18 bölge kan merkezinde üretilen ve imha edilen kan ürünlerinin miktarını ve imha nedenlerini ortaya ...
Yasemin Aslan, Beyza Yilmaz
doaj   +1 more source

Enrichment of plasma in platelets and extracellular vesicles by the counterflow to erythrocyte settling [PDF]

open access: bronze, 2021
Darja Božič   +9 more
openalex   +1 more source

Promiscuous stimulation of HSP70 ATPase activity by parasite‐derived J‐domains

open access: yesFEBS Open Bio, EarlyView.
The malaria parasite Plasmodium falciparum exports three highly homologous yet functionally divergent J‐domain proteins into human erythrocytes. Here, we show that J‐domains isolated from all three proteins effectively stimulate the ATPase activity of both endogenous host and exported parasite HSP70 chaperones.
Julian Barth   +6 more
wiley   +1 more source

Efficacy of Vitazal® in foals with anemia syndrome

open access: yesTheoretical and Applied Veterinary Medicine, 2020
Syndrome of anemia in foals manifests itself in the first weeks of their life, and occurs because of oxygen deficiency and excess of carbon dioxide after birth, and caused by a deficiency of trace minerals (iron, copper, cobalt, zinc, iodine, selenium ...
V. I. Holovakha   +4 more
doaj   +1 more source

A New Blood Diluent for Counting the Erythrocytes and Leucocytes of the Chicken

open access: yes, 1952
INTRODUCTION HEMATOLOGICAL studies on chickens have long been hampered by the lack of a quick, readily applicable, and quantitative method for the enumeration of the leucocytes.
M. P. Natt, C. A. Herrick
semanticscholar   +1 more source

Age Sensitivity of NFκB Abundance and Programmed Cell Death in Erythrocytes Induced by NFκB Inhibitors

open access: yesCellular Physiology and Biochemistry, 2013
Background/Aims: Erythrocytes may enter eryptosis, a suicidal death characterized by cell shrinkage and phosphatidylserine exposure at the erythrocyte outer membrane.
Mehrdad Ghashghaeinia   +11 more
semanticscholar   +1 more source

The Aging Blood: Cellular Origins, Circulating Drivers, and Therapeutic Potential

open access: yesAging and Cancer, EarlyView.
As a conduit linking all organs, the blood system both reflects and actively drives systemic aging. This review highlights how circulating pro‐aging and antiaging factors and age‐associated hematopoietic stem cell dysfunction contribute to immunosenescence and multi‐organ decline, positioning the hematopoietic system as a target for aging intervention.
Hanqing He, Jianwei Wang
wiley   +1 more source

Inhibition of Classical and Alternative Complement Pathway by Ravulizumab and Eculizumab

open access: yesAnnals of Clinical and Translational Neurology, EarlyView.
ABSTRACT Objective To explore the feasibility of classical (CH50) and alternative (AH50) complement pathway activity as potential biomarkers for treatment guidance and monitoring during therapy with ravulizumab in patients with generalized myasthenia gravis (gMG) and compare these to therapeutic drug monitoring under eculizumab.
Lea Gerischer   +14 more
wiley   +1 more source

Potential Modulation of Vascular Function by Nitric Oxide and Reactive Oxygen Species Released From Erythrocytes

open access: yesFrontiers in Physiology, 2018
The primary role for erythrocytes is oxygen transport that requires the reversible binding of oxygen to hemoglobin. There are, however, secondary reactions whereby the erythrocyte can generate reactive oxygen species (ROS) and nitric oxide (NO). ROS such
Joseph M. Rifkind   +6 more
doaj   +1 more source

Exploratory Analysis of ELP1 Expression in Whole Blood From Patients With Familial Dysautonomia

open access: yesAnnals of Clinical and Translational Neurology, EarlyView.
ABSTRACT Background Familial dysautonomia (FD) is a hereditary neurodevelopmental disorder caused by aberrant splicing of the ELP1 gene, leading to a tissue‐specific reduction in ELP1 protein expression. Preclinical models indicate that increasing ELP1 levels can mitigate disease manifestations.
Alejandra González‐Duarte   +13 more
wiley   +1 more source

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