DIEP reconstruction in a patient with Factor V Leiden deficiency: A case report and literature review. [PDF]
Quinn O, Bhatti S, Rosich-Medina A.
europepmc +3 more sources
Factor V Leiden Mutation and Neonatal Stroke
Three infants with familial factor V Leiden mutation and neonatal cerebrovascular disorders are reported from the Children’s Hospital of Philadelphia, PA.
J Gordon Millichap
doaj +2 more sources
Prothrombin G20210A and Factor V Leiden Variants Are Not Associated With Thrombotic Events in Congenital Heart Disease: An Observational Trial. [PDF]
Ladha FA +16 more
europepmc +2 more sources
Fator V de Leiden Factor V Leiden
José M. P. Godoy
doaj +2 more sources
Acute graft thrombosis in a patient with factor V Leiden mutation: A case report and review of literature. [PDF]
Lekehal B +7 more
europepmc +3 more sources
The prevalence of Factor V Leiden (Arg506Gln) mutation in King Khalid University Hospital patients, 2017–2019 [PDF]
Arg506Gln mutation is responsible for one of the procoagulant factors and most common inherited thrombophilia in the Factor V Leiden (FVL) family. The replacement of the missense mutation for Arg506Gln / R506Q is at 1691st position from Guanine to ...
Alotaibi, Abdulaziz A. +10 more
core +1 more source
A Rare Case of Quadruple Limb Amputation due to Factor V Leiden Mutation
Factor V Leiden, an autosomal dominant gene mutation that leads to increase risk of forming thrombophilia. Amputation in patients with Factor V leiden mutation is known but none describe quad-limb amputation in this setting.
Ann Noble Zachariah, Ravi Sankaran
doaj +1 more source
Left Renal Artery Thrombosis in a Patient With von Willebrand Disease Type 3 and Factor V Leiden Heterozygosity. [PDF]
Albright NL, Kamboj S, Lucas JM.
europepmc +3 more sources
Factor V Leiden and Inflammation [PDF]
Factor V Leiden, is a variant of human factor V (FV), also known as proaccelerin, which leads to a hypercoagulable state. Along these years, factor V Leiden (FVL) has been studied from the pathophysiologic point of view, and research has been focused on finding clinical approaches for the management of the FVL associated to a trombophilic state.
Silvia Perez-Pujol +2 more
openaire +2 more sources
Factor V Leiden, formed by a genetic mutation, disrupts the body's anticoagulation defense system. It was isolated as recently as 1994 as a risk factor for venous thromboembolism. Factor V Leiden has also been linked to preeclampsia, pregnancy loss, and fetal growth restriction.
Deborah L. Ornstein, Mary Cushman
openaire +3 more sources

