Results 211 to 220 of about 77,059 (283)

The factor V Leiden mutation may predispose women to severe preeclampsia.

American Journal of Obstetrics and Gynecology, 1996
D. Dizon-Townson, Lesa M. Nelson, K. L. Easton, K. Ward   +3 more
semanticscholar   +3 more sources

World distribution of factor V Leiden mutation

Lancet, 1996
Gou, D., Naipal, A., Reitsma, P. H.
exaly   +5 more sources

Factor V Leiden mutation and venous thrombosis

The Lancet, 1995
K.A. Davies, H. Ireland, P. Athanassiou, S. Loizou, D. Lane, M.J. Walport, Sinasi Özsoylu, Kiyohiko Hatake, Yasusada Miura   +8 more
openaire   +3 more sources

Selection pressure for the factor-V-Leiden mutation and embryo implantation.

Lancet, The, 2001
W. Göpel, M. Ludwig, Ann K Junge, T. Kohlmann, K. Diedrich, J. Möller   +5 more
semanticscholar   +3 more sources

Factor V Leiden mutation in Sneddon syndrome

Lupus, 2003
Sneddon syndrome (SNS) is characterizedby the associationof ischaemic cerebrovascularevents and widespread livedo racemosa. Its pathophysiology is still controversial. The aim of this study was to evaluate the prevalence of factor V Leiden mutation in consecutive patients referred for SNS according to antiphospholipid antibodies (aPL) status.
R, Besnier, C, Francès, A, Ankri, M, Aiach, J C, Piette   +4 more
openaire   +2 more sources

Factor V Leiden Mutation and Pregnancy-related Complications

Obstetric Anesthesia Digest, 2010
The objective of the study was to determine the prevalence of the factor V Leiden (FVL) mutation and its association with obstetric complications, blood loss during delivery, and venous thromboembolism (VTE).This was a prospective, observational, case-cohort study of 491 FVL carriers and 1055 controls derived from 6003 screened women.
Ulla, Kjellberg, Marianne, van Rooijen, Katarina, Bremme, Margareta, Hellgren   +3 more
openaire   +2 more sources

Homozygous factor V Leiden and double heterozygosity for factor V Leiden and prothrombin mutation

Journal of Thrombosis and Thrombolysis, 2012
The most common forms of familial thrombophilia are factor V Leiden (FVL) and prothrombin mutation (PTM). Homozygous FVL and PTM have long been feared conditions thought to cause high rates of morbidity and mortality. To analyse clinical features in patients with homozygous FVL and PTM, as well as patients with double heterozygosity for FVL and PTM ...
Ymir, Saemundsson, Signý Vala, Sveinsdottir, Henrik, Svantesson, Peter J, Svensson   +3 more
openaire   +2 more sources

Factor V Leiden Mutation and Budd-Chiari Syndrome

Blood, 1998
To the Editor: Budd-Chiari syndrome is characterized by the hepatic venous outflow obstruction. A multifactorial interaction between the genetic and circumstantial risk factors may be responsible for this kind of disorder.
D, Mohanty, S, Shetty, T S, Narayanan, P, Abraham   +3 more
openaire   +2 more sources

Factor V Leiden Mutation in Pregnancy

Journal of Obstetric, Gynecologic & Neonatal Nursing, 2004
Normal maternal adaptation to pregnancy significantly increases the risk for thrombus formation. Inherited thrombophilias further increase risk for deep venous thrombosis and adverse outcome in pregnancy. Factor V Leiden mutation is the most common inherited thrombophilia, occurring in approximately 5% of the White and 1% of the Black populations ...
openaire   +2 more sources

Factor V Leiden mutation: A nursing perspective

Journal of Vascular Nursing, 1998
The factor V Leiden mutation is a recently described autosomal dominant genetic risk factor for venous thromboembolism (VTE). Persons who are heterozygous or homozygous for this disorder are at 4 to 7 times and 50 to 100 times increased risk, respectively, for VTE.
openaire   +2 more sources