Results 81 to 90 of about 1,787,060 (299)

Predictors of response and rational combinations for the novel MCL‐1 inhibitor MIK665 in acute myeloid leukemia

open access: yesMolecular Oncology, EarlyView.
This study characterizes the responses of primary acute myeloid leukemia (AML) patient samples to the MCL‐1 inhibitor MIK665. The results revealed that monocytic differentiation is associated with MIK665 sensitivity. Conversely, elevated ABCB1 expression is a potential biomarker of resistance to the treatment, which can be overcome by the combination ...
Joseph Saad   +17 more
wiley   +1 more source

Peritonite meconial como diagnóstico diferencial de ascite fetal: relato de caso Meconium peritonitis in the differential diagnosis of fetal ascites: a case report

open access: yesRevista Brasileira de Ginecologia e Obstetrícia, 1999
Introdução: a peritonite meconial, como resultado da perfuração intestinal fetal, apresenta baixa incidência (1:30.000 nascimentos) e elevada mortalidade (em torno de 50%). Os achados ecográficos pré-natais incluem ascite e calcificações intra-abdominais.
Melania Maria Ramos de Amorim   +6 more
doaj   +1 more source

International estimated fetal weight standards of the INTERGROWTH‐21st Project

open access: yesUltrasound in Obstetrics and Gynecology, 2017
Estimated fetal weight (EFW) and fetal biometry are complementary measures used to screen for fetal growth disturbances. Our aim was to provide international EFW standards to complement the INTERGROWTH‐21st Fetal Growth Standards that are available for ...
J. Stirnemann   +244 more
semanticscholar   +1 more source

Characterizing epithelial‐mesenchymal transition‐linked heterogeneity in breast cancer circulating tumor cells at a single‐cell level

open access: yesMolecular Oncology, EarlyView.
In over 50% of non‐metastatic breast cancer patients, circulating tumor cells (CTCs) along the whole epithelial‐mesenchymal transition spectrum are detected. Total CTC number and individual phenotypes relate to aggressive disease characteristics, including lymph node involvement and higher tumor proliferation. At the single‐cell level, mesenchymal CTCs
Justyna Topa   +14 more
wiley   +1 more source

Editorial – Potential risks of the SARS-related coronavirus 2 infection [COVID-19] in pregnancy [PDF]

open access: yesCellR4, 2020
A. Sfakianaki   +5 more
doaj   +1 more source

Prevalence of erythrocyte alloimmunization in polytransfused patients [PDF]

open access: yesEinstein (São Paulo), 2011
Objective: To determine the incidence and the rate of red blood cellalloimmunization in polytransfused patients. Methods: A polytransfusedpatient was defined as having received at least 6 units of red cellconcentrates during a 3-month period. The records
Roberto de Oliveira Cruz   +6 more
doaj  

Deep learning-based parameter estimation in fetal diffusion-weighted MRI

open access: gold, 2021
Davood Karimi   +6 more
openalex   +1 more source

Glycosylated LGALS3BP is highly secreted by bladder cancer cells and represents a novel urinary disease biomarker

open access: yesMolecular Oncology, EarlyView.
Urinary LGALS3BP is elevated in bladder cancer patients compared to healthy controls as detected by the 1959 antibody–based ELISA. The antibody shows enhanced reactivity to the high‐mannose glycosylated variant secreted by cancer cells treated with kifunensine (KIF).
Asia Pece   +18 more
wiley   +1 more source

Fetal vascular malperfusion, an update

open access: yesActa Pathologica, Microbiologica et Immunologica Scandinavica (APMIS), 2018
Fetal vascular malperfusion is the most recent term applied to a group of placental lesions indicating reduced or absent perfusion of the villous parenchyma by the fetus.
R. Redline, Sanjita Ravishankar
semanticscholar   +1 more source

Survivin and Aurora Kinase A control cell fate decisions during mitosis

open access: yesMolecular Oncology, EarlyView.
Aurora A interacts with survivin during mitosis and regulates its centromeric role. Loss of Aurora A activity mislocalises survivin, the CPC and BubR1, leading to disruption of the spindle checkpoint and triggering premature mitotic exit, which we refer to as ‘mitotic slippage’.
Hana Abdelkabir   +2 more
wiley   +1 more source

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