Results 111 to 120 of about 346,213 (330)
HIV protease inhibitors against the viral protease are often hampered by drug resistance mutations in protease and in the viral substrate Gag. To overcome this drug resistance and inhibit viral maturation, targeting Gag alongside protease rather than ...
Chinh Tran-To Su +2 more
doaj +1 more source
Live-cell observation of cytosolic HIV-1 assembly onset reveals RNA-interacting Gag oligomers [PDF]
Jelle Hendrix +8 more
openalex +1 more source
Different methods for management of gaging reflex during prosthetic treatment
Tsvetelina Kanlieva +2 more
openalex +2 more sources
Merging 4D printing with magneto‐responsive shape memory polymers opens new avenues for intelligent, reconfigurable systems. This review navigates cutting‐edge fabrication techniques, magnetic fillers, and smart polymer matrices, unveiling their potential in soft robotics, biomedical devices, and wearable tech.
Kiandokht Mirasadi +7 more
wiley +1 more source
Susceptibility of Human Immunodeficiency Virus Type 1 to the Maturation Inhibitor Bevirimat Is Modulated by Baseline Polymorphisms in Gag Spacer Peptide 1 [PDF]
Kurt Van Baelen +9 more
openalex +1 more source
Human participants with early‐ or chronic‐stage tendinopathy and healthy controls are investigated using ultrasound Doppler, 3T and 7T MRI, and tendon biopsies. Tendon swelling and hyperperfusion are evident early, while vascular growth and extracellular matrix changes are most pronounced in chronic tendinopathy. Early intervention may provide superior
Max F. R. Merkel +11 more
wiley +1 more source
La notion de gag n'appartient pas en propre à la bande dessinée. Il y a des gags au cinéma, ils caractérisent même un genre de cinéma, le cinéma burlesque. Il y a des gags à la télévision, pensez aux « vidéo-gags ». Il y a, depuis beaucoup plus longtemps, des gags au théâtre et dans tous les arts vivants qui mettent en scène le corps d'un acteur, le ...
openaire +4 more sources
In-vivo mutation rates and fitness landscape of HIV-1
Mutation rates and fitness costs of deleterious mutations are difficult to measure in vivo but essential for a quantitative understanding of evolution. Using whole genome deep sequencing data from longitudinal samples during untreated HIV-1 infection, we
Albert, Jan +4 more
core +1 more source
5’tRF‐GlyGCC promotes breast cancer malignancy by binding to LDHA. This interaction, facilitated by FGFR1 and LDHA phosphorylation, enhances glycolysis. Additionally, 5’tRF‐GlyGCC/LDHA signaling recruits and polarizes macrophages into a pro‐tumor M2 state via CCL7, thus remodeling the tumor microenvironment.
Cheng Yi +17 more
wiley +1 more source

