Results 161 to 170 of about 14,424 (294)
Diversity and enzymatic activity of Polish beehive products microbiota, and characterization of a novel β-galactosidase from Paenibacillus sp. 8. [PDF]
Rosińska A+3 more
europepmc +1 more source
HYDROLASE ACTIVITIES IN BRAIN OF NEUROLOGICAL MUTANTS: CEREBROSIDE GALACTOSIDASE, NITROPHENYL GALACTOSIDE HYDROLASE, NITROPHENYL LUCOSIDE HYDROLASE AND SULPHATASE1 [PDF]
D. M. Bowen, Norman S. Radin
openalex +1 more source
This study reveals that in vitro senescence induction methods significantly influence intracellular biomolecular profiles, showing both shared and unique metabolic responses. A novel bioanalytical pipeline was developed to explore these subphenotypes, highlighting the importance of targeting metabolic redox circuits and serine/taurine pathways for ...
Domenica Berardi+11 more
wiley +1 more source
Immobilized β-galactosidase BgaC from Bifidobacterium adolescentis retains stability and activity during repeated cycles of use. [PDF]
Mulualem DM+4 more
europepmc +1 more source
Chronic NAD depletion in fibroblasts lowers NAD levels, increases mitochondrial mass, and triggers an interferon‐dependent inflammatory response via mtDNA leakage through VDAC1, activating the cGAS‐STING pathway. This links NAD deficiency to metabolic and inflammatory processes, with implications for diseases associated with NAD decline.
Claudia C. S. Chini+14 more
wiley +1 more source
Beta-galactosidase: orientation and the carboxyl-terminal coding site in the gene. [PDF]
Jennifer Brown, D.M. Brown, Irving Zabin
openalex +1 more source
As a natural senomorphic agent, PQQ targets the intracellular molecule. HSPA8 of senescent cells, resulting in significantly reduced expression of the SASP. PQQ holds prominent potential to delay chronological aging and ameliorate age‐related conditions by acting as an effective senomorphic agent.
Birong Jiang+6 more
wiley +1 more source
Some Enzymic Properties of a Microbial beta-Galactosidase of Human Whole Saliva. [PDF]
Kauko K. Mäkinen+3 more
openalex +1 more source
Virus‐Induced Cellular Senescence Causes Pulmonary Sequelae Post‐Influenza Infection
Genetic or pharmacological depletion of senescent cells during influenza A virus infection led to complete recovery of the airway epithelium, indicating a marked acceleration of the epithelial repair process. Targeting senescent cells may represent a new preventive therapeutic option to reduce post‐influenza chronic lung damage.
Larissa Lipskaia+20 more
wiley +1 more source