Results 271 to 280 of about 64,020 (335)
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Cholecystokinin and gastric inhibitory polypeptide not glucagonotropic in dogs

Journal of Surgical Research, 1980
Abstract The role of cholecystokinin (CCK) and gastric inhibitory polypeptide (GIP) as stimulators of glucagon secretion under physiologic conditions is uncertain. Previous studies have utilized CCK preparations now known to have been contaminated with GIP and possibly other gastrointestinal peptides.
K R, Sirinek   +3 more
openaire   +2 more sources

Gastric Inhibitory Polypeptide (GIP)

1988
Publisher Summary This chapter describes two apparently unrelated physiological phenomena for a single peptide that is gastric inhibitory polypeptide (GIP). These physiological actions are the inhibition of gastric acid secretion and stimulation of insulin release. GIP is a major hormone involved in the gastrointestinal regulation of insulin release,
J.C. Brown   +2 more
openaire   +1 more source

Determinants of gastric inhibitory polypeptide and insulin secretion

Metabolism, 1981
The plasma concentrations of gastric inhibitory polypeptide (IR-GIP), insulin, and glucose were measured in 5 dogs after administration of various constituents of food. IR-GIP responses produced by galactose and fructose were minimal compared to those of glucose.
R H, Williams, J M, May, J B, Biesbroeck
openaire   +2 more sources

Gastric Inhibitory Polypeptide: the neglected incretin revisited

Regulatory Peptides, 2002
After the ingestion of fat- and glucose-rich meals, gut hormones are secreted into the circulation in order to stimulate insulin secretion. This so-called "incretin effect" is primarily conferred by Glucagon-like peptide 1 (GLP-1) and Gastric Inhibitory Polypeptide (GIP).
Juris J, Meier   +3 more
openaire   +2 more sources

Gastric Inhibitory Polypeptide

Archives of Surgery, 1974
Gastric inhibitory polypeptide (GIP), immunoreactive insulin (IRI), and glucose levels in response to orally administered glucose were determined in ten patients with vagotomy and pyloroplasty (V&P) and in 21 normal subjects. The V&P patients had the expected early and exaggerated increase in mean serum glucose with a subsequent decline to a level ...
N R, Thomford   +4 more
openaire   +2 more sources

The evaluation of gastric inhibitory polypeptide (GIP) as the enterogastrone

Journal of Surgical Research, 1979
In 1886 Ewald and Boas [4] first showed that olive oil added to a test meal of starch paste given to human subjects inhibited both gastric emptying and acid secretion. Pavlov and his pupil Babkin (1928) later showed that this effect was of duodenal rather than of gastric origin.
P, Soon-Shiong, H T, Debas, J C, Brown
openaire   +2 more sources

Changes in Gastric Inhibitory Polypeptide (GIP) After Roux-en-Y Gastric Bypass in Obese Patients: a Meta-analysis

Obesity Surgery, 2022
Zhiguang Gao   +6 more
semanticscholar   +1 more source

Gastric Inhibitory Polypeptide (GIP) in Chronic Pancreatitis

The Journal of Clinical Endocrinology & Metabolism, 1976
The plasma gastric inhibitory polypeptide (GIP), pancreatic glucagon-like immunoreactivity (PGLI), and gut glucagon-like immunoreactivity (GGLI) responses to oral glucose have been measured in five patients with chronic pancreatitis (with diabetic glucose tolerance tests) and in matched nondiabetic controls. Plasma GIP levels rise rapidly after glucose
J L, Botha, A I, Vinik, J C, Brown
openaire   +2 more sources

Glucagon, gastric inhibitory polypeptide and the gastrocolic response

The American Journal of Surgery, 1982
Serosal bipolar electrodes and strain gauge force transducers were placed on the right and left colon in subhuman primates to record spike discharges and circular muscular contractions. The effect of glucagon on colonic motor and electrical activity were studied before and after meals.
J A, Strom   +4 more
openaire   +2 more sources

Gastric Inhibitory Polypeptide in Obesity and Diabetes Mellitus*

The Journal of Clinical Endocrinology & Metabolism, 1984
Gastric inhibitory polypeptide (GIP) concentrations may be influenced by obesity, diabetes, and glucagon deficiency and be under feedback inhibition by insulin. To assess these factors, insulin-dependent diabetic, totally pancreatectomized diabetic, and lean and obese noninsulin-dependent diabetic patients were studied twice, once during partial ...
F J, Service   +5 more
openaire   +2 more sources

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