Upregulation of senescence-associated gene expression levels in human gingival tissue affected by periodontitis. [PDF]
Kim SJ +7 more
europepmc +1 more source
Evaluation of IL-4, MIP-1α, and MMP-9 gene expression levels in peri-implant tissues in peri-implantitis. [PDF]
Giro G +5 more
europepmc +1 more source
Etoposide induces DNA damage, activating p53‐dependent apoptosis via caspase‐3/7, which cleaves PARP1. Dammarenediol II enhances this apoptotic pathway by suppressing O‐GlcNAc transferase activity, further decreasing O‐GlcNAcylation. The reduction in O‐GlcNAc levels boosts p53‐driven apoptosis and influences the Akt/GSK3β/mTOR signaling pathway ...
Jaehoon Lee +8 more
wiley +1 more source
In Silico Characterisation and Determination of Gene Expression Levels of the CPK Family Under Saline Stress Conditions in Chenopodium quinoa Willd. [PDF]
Lima-Huanca L +3 more
europepmc +1 more source
Analysis of ADORA2A, MTA1, PTGDS, PTGS2, NSF, and HNMT Gene Expression Levels in Peripheral Blood of Patients with Early Stages of Parkinson's Disease. [PDF]
Semenova EI +13 more
europepmc +1 more source
Chromatin-level Analysis of Regulation of Gene Expression
M, Harata, S, Mizuno
openaire +3 more sources
Aptamers are used both therapeutically and as targeting agents in cancer treatment. We developed an aptamer‐targeted PLGA–TRAIL nanosystem that exhibited superior therapeutic efficacy in NOD/SCID breast cancer models. This nanosystem represents a novel biotechnological drug candidate for suppressing resistance development in breast cancer.
Gulen Melike Demirbolat +8 more
wiley +1 more source
Assessment of TSPO Gene Expression Levels in Colorectal Cancer Tumors: A Paired Sample Study. [PDF]
Karami Gorji H +9 more
europepmc +1 more source
Diverse environmental perturbations reveal the evolution and context-dependency of genetic effects on gene expression levels. [PDF]
Lea AJ, Peng J, Ayroles JF.
europepmc +1 more source
Targeting p38α in cancer: challenges, opportunities, and emerging strategies
p38α normally regulates cellular stress responses and homeostasis and suppresses malignant transformation. In cancer, however, p38α is co‐opted to drive context‐dependent proliferation and dissemination. p38α also supports key functions in cells of the tumor microenvironment, including fibroblasts, myeloid cells, and T lymphocytes.
Angel R. Nebreda
wiley +1 more source

