Results 61 to 70 of about 1,401 (195)

Gilbert's syndrome and the risk of death: a population-based cohort study [PDF]

open access: yes, 2013
Gilbert's syndrome is a common familial hyperbilirubinemia that may reduce the risk of various age-related diseases because of the antioxidant properties of bilirubin.
Pereira, SP   +3 more
core  

GILBERT’S SYNDROME IN CHILDREN: CONTEMPORARY DIAGNOSTIC POTENTIALITIES

open access: yesПедиатрическая фармакология, 2011
Gilbert’s syndrome is a benign indirect hyperbilirubinemia of the hereditary nature, caused by deficiency of the enzyme uridindiphosphatglucuronitransferase. UGT1A1 gene is localized on chromosome 2q37.
I.N. Zakharova   +7 more
doaj   +2 more sources

Talking therapies for anxiety and depression in people affected by dementia: A scoping meta‐review

open access: yesPsychology and Psychotherapy: Theory, Research and Practice, EarlyView.
Abstract Background Symptoms of anxiety and depression are common in people affected by dementia (people with dementia and family carers). There are many reviews of psychological interventions to reduce stress and distress in people with dementia and family carers, but little cross‐fertilisation with the evidence base underpinning NHS Talking Therapies
Georgina Charlesworth   +2 more
wiley   +1 more source

Contribution of two missense mutations (G71R and Y486D) of the bilirubin UDP glycosyltransferase (UGT1A1) gene to phenotypes of Gilbert's syndrome and Crigler–Najjar syndrome type II

open access: yes, 1998
In our mutation analyses of bilirubin UDP glycosyltransferase (UGT1A1) gene, we encountered six patients with Crigler–Najjar syndrome type II who were double homozygotes for G71R and Y486D, a patient with Gilbert's syndrome who was a single homozygote ...
Yamamoto, Kazuo   +4 more
core   +1 more source

Women in space: A review of known physiological adaptations and health perspectives

open access: yesExperimental Physiology, EarlyView.
Abstract Exposure to the spaceflight environment causes adaptations in most human physiological systems, many of which are thought to affect women differently from men. Since only 11.5% of astronauts worldwide have been female, these issues are largely understudied.
Millie Hughes‐Fulford   +4 more
wiley   +1 more source

Hyperbilirubinemia following lenalidomide administration

open access: yesClinical Case Reports, 2018
Key Clinical Message Asymptomatic hyperbilirubinemia in a patient with no underlying liver disease or renal impairment while on lenalidomide therapy may be attributable to the unmasking of previously undiagnosed Gilbert's syndrome, as previously shown in
Veronica Azmy, Natalia Neparidze
doaj   +1 more source

Insights from animal models: Dissecting the independent roles of oxygen and nutrients in the fetal origins of cardiovascular disease

open access: yesThe Journal of Physiology, EarlyView.
Abstract figure legend This review utilized animal models of complicated human pregnancies that result in reduced fetal nutrient or oxygen delivery, or combined nutrient and oxygen delivery, to elucidate their independent and/or synergistic contributions to the development of high‐risk cardiac phenotypes.
Melanie R. Bertossa   +8 more
wiley   +1 more source

Coexisting Duchenne Muscular Dystrophy and Gilbert's Syndrome: A Case Report

open access: yes, 2013
Gilbert's sendromu konjuge olmayan hiperbilirubinemi ile karekterize bir hastalıktır. 5 yaşında erkek çocuğu hafif sarılık ile hastanemize getirildi.
Şakir Altunbaşak   +4 more
core  

Genetic variation underlying common hereditary hyperbilirubinaemia (Gilbert's syndrome) and respiratory health in the 1946 British birth cohort [PDF]

open access: yes, 2014
BACKGROUND & AIMS: Bilirubin has potent antioxidant properties in vitro and raised serum levels have been associated with lower rates of respiratory disease.
Swallow, DM   +9 more
core   +1 more source

Prevalence of suspected Gilbert’s syndrome in Golestan province, northern Iran (2014)

open access: yesمجله دانشگاه علوم پزشکی گرگان, 2021
Background and Objective: Gilbertchr('39')s syndrome is a relatively common genetic disorder, which is caused by defection in uridine diphosphate glucuronosyl transferase enzyme. The indirect bilirubin increases in this syndrome, although the function of
Azadeh Aliarab   +7 more
doaj  

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