Results 301 to 310 of about 365,436 (354)
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Trypanosome glucose transporters
Molecular and Biochemical Parasitology, 1998, by contrast adopts an intracellularenvironment within their mammalian hosts. Inva-sion and passage through different anatomicallocations within insect vectors also distinguishesthe parasites.All trypanosome species use glucose as a crucialsource of energy, and all have specific plasmamembrane transporters to facilitate the uptake ofthis molecule. Four
M P, Barrett +4 more
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Glucose Transporters in the Transepithelial Transport of Glucose
Journal of Electron Microscopy, 1996Glucose transporters are integral membrane proteins that mediate the transport of glucose and structurally-related substances across the cellular membranes. Two families of glucose transporter have been identified: the facilitated-diffusion glucose transporter family (GLUT family), and the NA(+)-dependent glucose transporter one (SGLT family).
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Glucose transport in lymphocytes
Pflügers Archiv - European Journal of Physiology, 2020Glucose uptake into lymphocytes is accomplished by non-concentrative glucose carriers of the GLUT family (GLUT1, GLUT3, GLUT4, GLUT6) and/or by the Na+-coupled glucose carrier SGLT1. The latter accumulates glucose against glucose gradients and is still effective at very low extracellular glucose concentrations.
Florian Lang +6 more
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Glucose transport and apoptosis
Apoptosis, 2000The transport and metabolism of glucose modify programmed cell death in a number of different cell types. This review presents three cell death paradigms that link a decrease in glucose transport to apoptosis. Although these pathways overlap, the glucose-dependent stimuli that trigger cell death differ.
K H, Moley, M M, Mueckler
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2011
Glucose is obtained directly from the diet, principally following the hydrolysis of ingested disaccharides and polysaccharides, and by the synthesis from other substrates in organs such as the liver. Glucose derived from the diet is transferred from the lumen of the small intestine, and both dietary glucose and glucose synthesized within the body have ...
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Glucose is obtained directly from the diet, principally following the hydrolysis of ingested disaccharides and polysaccharides, and by the synthesis from other substrates in organs such as the liver. Glucose derived from the diet is transferred from the lumen of the small intestine, and both dietary glucose and glucose synthesized within the body have ...
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Nihon rinsho. Japanese journal of clinical medicine, 1997
All mammalian cells contain trans-membrane transport systems for the stereo specific uptake of glucose. These systems are divided into two groups, a facilitated diffusion system driven by the concentration differences across the membrane and an energy-dependent, active transport system.
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All mammalian cells contain trans-membrane transport systems for the stereo specific uptake of glucose. These systems are divided into two groups, a facilitated diffusion system driven by the concentration differences across the membrane and an energy-dependent, active transport system.
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Glucose Transport and Glucose Transporters in Muscle and Their Metabolic Regulation
Diabetes Care, 1990Skeletal muscle is the primary tissue responsible for insulin-dependent glucose uptake in vivo; therefore, glucose uptake by this tissue plays an important role in determining glycemia. Glucose uptake in muscle occurs by a system of facilitated diffusion involving at least two distinct glucose transporters, GLUT-1 and GLUT-4.
A, Klip, M R, Pâquet
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Glucose transporter protein syndromes
2002Publisher Summary The disturbances of carbohydrate homeostasis have interested clinicians and scientists since the recognition of diabetes mellitus. A parallel line of study emerged in the study of patients with recurrent or chronic hypoglycemic syndromes.
Darryl C, De Vivo +3 more
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Diabetes Care, 1992
Three major metabolic abnormalities contribute to hyperglycemia in non-insulin-dependent diabetes mellitus (NIDDM) including defective glucose-induced insulin secretion, elevated rates of hepatic glucose output, and insulin's impaired ability to stimulate glucose uptake in peripheral target tissues (insulin resistance). These functions involve cellular
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Three major metabolic abnormalities contribute to hyperglycemia in non-insulin-dependent diabetes mellitus (NIDDM) including defective glucose-induced insulin secretion, elevated rates of hepatic glucose output, and insulin's impaired ability to stimulate glucose uptake in peripheral target tissues (insulin resistance). These functions involve cellular
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