Results 191 to 200 of about 192,172 (285)
GM-CSF Armed Oncolytic Adenovirus Enhances T-Cell Infiltration and Suppresses Local and Distal Tumor Growth. [PDF]
Xu HW +10 more
europepmc +1 more source
RORγt‐APCs: The New Masters of Oral Tolerance
ABSTRACT Oral tolerance is defined by the hypo‐responsiveness of our body to fed antigens, and its failure can lead to immune‐mediated diseases, such as allergy, chronic inflammation and autoimmune diseases. Decades of research have demonstrated that antigen‐presenting cells (APCs) promote oral tolerance by inducing regulatory T cells (Tregs) and/or ...
Thierry Gauthier, WanJun Chen
wiley +1 more source
Adjunctive GM-CSF therapy enhances host defense against systemic <i>Candida auris</i> infection in immunosuppressed mice. [PDF]
Mattos EC +5 more
europepmc +1 more source
Fibroblast Plasticity in Inflammation of the Barrier
Under inflammatory condition, fibroblasts in barrier tissues integrate inflammatory, senescent, and mechanical inputs. These pathways activate central signaling networks to drive fibroblast phenotypic transition, enabling cytokine production, immune cell recruitment, and extracellular matrix remodeling.
Chen Zhang, Hao Wu, Wei Li
wiley +1 more source
The power of GM-CSF: immune regulation in the defense against <i>Phialophora verrucosa</i> infection. [PDF]
Dong Q +5 more
europepmc +1 more source
Abstract Background CD163 is a scavenger receptor predominantly expressed on the surfaces of macrophages in various mammalian species and is a marker of anti‐inflammatory (M2‐like) macrophages. High density of CD163‐positive tumor‐associated macrophages (TAMs) is associated with worse prognosis in various patient tumors.
Yoichi Saito +9 more
wiley +1 more source
mGem: When immunity turns against itself-GM-CSF autoantibodies drive opportunistic infection risk. [PDF]
Sengupta S, Williamson PR.
europepmc +1 more source
Inhaled GM-CSF for Pulmonary Alveolar Proteinosis
Papiris, S.A., Griese, M., Manali, E.D.
openaire +3 more sources
Abstract Aims The extent of irreversible cardiomyocyte necrosis after acute myocardial infarction (AMI) is a major determinant of residual left ventricular (LV) function and clinical outcome. Cell therapy based on CD34+ cells has emerged as an option to help repair the myocardium and to improve outcomes.
Jerome Roncalli +17 more
wiley +1 more source
Efficacy and safety of entecavir, peginterferon alfa-2b and GM-CSF combination therapy: the anchor randomized controlled trial. [PDF]
Wu D +22 more
europepmc +1 more source

