Results 31 to 40 of about 5,470,060 (264)

Morbidity After Total Thyroidectomy for Benign Thyroid Disease: Comparison of Graves' Disease and Non-Graves' Disease

open access: yesKaohsiung Journal of Medical Sciences, 2006
The purpose of this study was to review the safety of total thyroidectomies for benign thyroid disease, with special emphasis on the comparison between Graves' disease and non-Graves' disease.
Feng-Yu Chiang   +6 more
doaj   +1 more source

Carcinomas in the remnant thyroid after thyroidectomy for Graves' disease: report of a case [PDF]

open access: yes, 2013
A 52-year-old female who had undergone subtotal thyroidectomy for Graves' disease 20 years previously was referred to us with nodules in both lobes in her remnant thyroid.
Minami, Shigeki   +8 more
core  

Diversity and complexity in neural organoids

open access: yesFEBS Letters, EarlyView.
Neural organoid research aims to expand genetic diversity on one side and increase tissue complexity on the other. Chimeroids integrate multiple donor genomes within single organoids. Self‐organising multi‐identity organoids, exogenous cell seeding, or enforced assembly of region‐specific organoids contribute to tissue complexity.
Ilaria Chiaradia, Madeline A. Lancaster
wiley   +1 more source

Riedel Thyroiditis in a Patient with Graves Disease [PDF]

open access: yesEndocrinology and Metabolism, 2013
Riedel's thyroiditis is a rare form of infiltrative and inflammatory disease of the thyroid gland and can be associated with systemic fibrotic processes, Hashimoto thyroiditis and Graves disease.
Doo Young Lee   +4 more
doaj   +1 more source

Structure‐forward targeting of claudins with synthetic binders

open access: yesFEBS Letters, EarlyView.
Claudins form the paracellular barriers between epithelial and endothelial tissues at tight junctions and are targets for molecular binders with the goal of modulating barrier permeability. Claudin‐binding molecules are relevant in drug delivery or in altering claudin interactions with disease‐causing proteins.
Alex J. Vecchio
wiley   +1 more source

Evaluation of vitamin D status and its impact on thyroid related parameters in new onset Graves' disease- A cross-sectional observational study

open access: yesIndian Journal of Endocrinology and Metabolism, 2019
Aims and Objective: We aimed to compare serum vitamin D level in new onset Graves' disease versus age and sex matched controls. Furthermore, we assessed the correlation of vitamin D with hormonal parameters and antibody titers in Graves' disease ...
Swayamsidha Mangaraj   +5 more
doaj   +1 more source

The ubiquitin system in normal and infected germinal center B cells

open access: yesFEBS Letters, EarlyView.
The ubiquitin system plays a central role in germinal center (GC) B cells, influencing differentiation to long‐lived memory B cells. Oncogenic gammaherpesviruses gain access to memory B cells by establishing latency in GC B cells. Mapping ubiquitin mechanisms in normal and infected GC B cells will define specific molecular circuits in B cells germane ...
Destiny Davis   +2 more
wiley   +1 more source

GRAVES' DISEASE [PDF]

open access: yesThe Journal of Nervous and Mental Disease, 1896
n ...
openaire   +2 more sources

How structural disorder and polyelectrolyte properties of prothymosin α enable its large interactome

open access: yesFEBS Letters, EarlyView.
In this review, complexes of the fully disordered and highly negatively charged prothymosin α are collected, described, and discussed. Linked to its poly‐electrostatic character, prothymosin α engages a large interactome through multiple different binding modes ranging from fully disordered complexes to short linear motif (SLiM)‐driven interactions ...
Ketty C. Tamburrini   +3 more
wiley   +1 more source

Catatony in Graves' disease

open access: yesEuropean Psychiatry, 1998
SummaryWe report a 27-year-old female patient with prior history of Graves' disease and relapsing episodes of tachycardia, hyperpyrexia, muscular rigidity and coma. With the subsequent manifestation of an acute schizophreniform psychotic disorder unresponsive to neuroleptics, the primary syndrome was re-classified as febrile catatony.
J, Rudolf   +3 more
openaire   +2 more sources

Home - About - Disclaimer - Privacy