Results 11 to 20 of about 359,357 (309)

Genome-Wide CRISPR-Cas9 Screen Does Not Identify Host Factors Modulating Streptococcus agalactiae β-Hemolysin/Cytolysin-Induced Cell Death

open access: yesMicrobiology Spectrum, 2022
Pore-forming toxins (PFTs) are commonly produced by pathogenic bacteria, and understanding them is key to the development of virulence-targeted therapies. Streptococcus agalactiae, or group B Streptococcus (GBS), produces several factors that enhance its
Ifrah Shahi   +4 more
doaj   +1 more source

Group B Streptococcus: Virulence Factors and Pathogenic Mechanism

open access: yesMicroorganisms, 2022
Group B Streptococcus (GBS) or Streptococcus agalactiae is a major cause of neonatal mortality. When colonizing the lower genital tract of pregnant women, GBS may cause premature birth and stillbirth.
Yuxin Liu, Jinhui Liu
doaj   +1 more source

Prevention of Perinatal Group B Streptococcus Infections

open access: yesMaternal-Fetal Medicine, 2020
. Group B streptococcus (GBS) is a leading cause of neonatal infection. Maternal vaginal-rectal colonization with GBS during the intrapartum period is a prerequisite for GBS early-onset disease (EOD).
Shang-Rong Fan   +5 more
doaj   +1 more source

Garlic: An Alternative Treatment for Group B Streptococcus

open access: yesMicrobiology Spectrum, 2021
Prenatal screening in pregnant women between 35 and 37 weeks of gestation and intrapartum antibiotic prophylaxis has successfully reduced the incidence of neonatal morbidity and mortality related to Streptococcus agalactiae.
Kátia Andrea de Menezes Torres   +4 more
doaj   +1 more source

Developing a serocorrelate of protection against invasive group B streptococcus disease in pregnant women: a feasibility study. [PDF]

open access: yes, 2019
BACKGROUND: Group B streptococcus is the leading cause of infection in infants. Currently, intrapartum antibiotic prophylaxis is the major strategy to prevent invasive group B streptococcus disease.
Andrews, N   +16 more
core   +1 more source

Adaptive response of neonatal sepsis-derived Group B Streptococcus to bilirubin [PDF]

open access: yes, 2018
This work was funded by the Neonatal Unit Endowment Fund, Aberdeen Maternity Hospital. RH is funded by a career researcher fellowship from NHS Research Scotland. SG was funded by the MRC Flagship PhD programme.
Berry, Susan   +10 more
core   +4 more sources

Association between genotypic diversity and biofilm production in group B Streptococcus [PDF]

open access: yes, 2016
Background: Group B Streptococcus (GBS) is a leading cause of sepsis and meningitis and an important factor in premature and stillbirths. Biofilm production has been suggested to be important for GBS pathogenesis alongside many other elements ...
Davies, H. Dele   +5 more
core   +2 more sources

Group B Streptococcus Colonization in Pregnancy

open access: yesSri Lankan Journal of Infectious Diseases, 2015
Introduction:  Group B β haemolytic streptococcus (GBS) is a colonizer of the female genital tract and a known cause of neonatal infections. Identification of GBS colonization in pregnancy is the key to prevent such infections.
BN Dissanayake, GC Herath, TM Gamage
doaj   +1 more source

Group B streptococcus cystitis presenting in a diabetic patient with a massive abdominopelvic abscess: a case report [PDF]

open access: yes, 2012
INTRODUCTION: Streptococcus agalactiae or group B streptococcus is a Gram-positive pathogen that is typically associated with neonatal disease and infection in pregnant women.
Chee K Tan   +4 more
core   +1 more source

Group A, B, C, and G Streptococcus Lancefield antigen biosynthesis is initiated by a conserved α-D-GlcNAc-β-1,4-L-rhamnosyltransferase [PDF]

open access: yes, 2019
Group A carbohydrate (GAC) is a bacterial peptidoglycan-anchored surface rhamnose polysaccharide (RhaPS) that is essential for growth of Streptococcus pyogenes and contributes to its ability to infect the human host.
Albesa-Jové   +110 more
core   +2 more sources

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