Results 11 to 20 of about 359,367 (303)

Genome-Wide CRISPR-Cas9 Screen Does Not Identify Host Factors Modulating Streptococcus agalactiae β-Hemolysin/Cytolysin-Induced Cell Death

open access: yesMicrobiology Spectrum, 2022
Pore-forming toxins (PFTs) are commonly produced by pathogenic bacteria, and understanding them is key to the development of virulence-targeted therapies. Streptococcus agalactiae, or group B Streptococcus (GBS), produces several factors that enhance its
Ifrah Shahi   +4 more
doaj   +1 more source

Prevalence and factors associated with group B streptococcal colonization in pregnant women [PDF]

open access: yesRevista Brasileira de Saúde Materno Infantil, 2021
Objectives: to estimate the prevalence and the factors associated with the colonization by group B streptococcus (GBS) in pregnant women from the urban area attended at health units in a municipality in northeastern Brazil.
Taís Viana Lédo de Oliveira   +3 more
doaj   +3 more sources

Prevention of Perinatal Group B Streptococcus Infections

open access: yesMaternal-Fetal Medicine, 2020
. Group B streptococcus (GBS) is a leading cause of neonatal infection. Maternal vaginal-rectal colonization with GBS during the intrapartum period is a prerequisite for GBS early-onset disease (EOD).
Shang-Rong Fan   +5 more
doaj   +1 more source

Group B Streptococcus: Virulence Factors and Pathogenic Mechanism

open access: yesMicroorganisms, 2022
Group B Streptococcus (GBS) or Streptococcus agalactiae is a major cause of neonatal mortality. When colonizing the lower genital tract of pregnant women, GBS may cause premature birth and stillbirth.
Yuxin Liu, Jinhui Liu
doaj   +1 more source

Garlic: An Alternative Treatment for Group B Streptococcus

open access: yesMicrobiology Spectrum, 2021
Prenatal screening in pregnant women between 35 and 37 weeks of gestation and intrapartum antibiotic prophylaxis has successfully reduced the incidence of neonatal morbidity and mortality related to Streptococcus agalactiae.
Kátia Andrea de Menezes Torres   +4 more
doaj   +1 more source

Developing a serocorrelate of protection against invasive group B streptococcus disease in pregnant women: a feasibility study. [PDF]

open access: yes, 2019
BACKGROUND: Group B streptococcus is the leading cause of infection in infants. Currently, intrapartum antibiotic prophylaxis is the major strategy to prevent invasive group B streptococcus disease.
Andrews, N   +16 more
core   +1 more source

Association between genotypic diversity and biofilm production in group B Streptococcus [PDF]

open access: yes, 2016
Background: Group B Streptococcus (GBS) is a leading cause of sepsis and meningitis and an important factor in premature and stillbirths. Biofilm production has been suggested to be important for GBS pathogenesis alongside many other elements ...
Davies, H. Dele   +5 more
core   +2 more sources

Group B Streptococcus Colonization in Pregnancy

open access: yesSri Lankan Journal of Infectious Diseases, 2015
Introduction:  Group B β haemolytic streptococcus (GBS) is a colonizer of the female genital tract and a known cause of neonatal infections. Identification of GBS colonization in pregnancy is the key to prevent such infections.
BN Dissanayake, GC Herath, TM Gamage
doaj   +1 more source

Preterm infants have deficient monocyte and lymphocyte cytokine responses to Group B Streptococcus [PDF]

open access: yes, 2011
Group B streptococcus GBS) is an important cause of early-and late-onset sepsis in the newborn. Preterm infants have markedly increased susceptibility and worse outcomes, but their immunological responses to GBS are poorly defined.
Currie, A.J.   +9 more
core   +2 more sources

Conjugative transfer of ICESde3396 between three β-hemolytic streptococcal species [PDF]

open access: yes, 2014
Background: Integrative conjugative elements (ICEs) are mobile genetic elements (MGEs) that possess all genes necessary for excision, transfer and integration into recipient genome. They also carry accessory genes that impart new phenotypic features to
Bauer, Michelle J.   +6 more
core   +1 more source

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