Results 201 to 210 of about 246,437 (319)

Mitochondrial targets in ischaemic heart disease and heart failure, and their potential for a more efficient clinical translation. A scientific statement of the ESC Working Group on Cellular Biology of the Heart and the ESC Working Group on Myocardial Function

open access: yesEuropean Journal of Heart Failure, EarlyView.
Improving clinical translation of mitoprotective therapies. 31P MRI, phosphorus‐31 magnetic resonance imaging; hiPSC‐CM, human‐induced pluripotent stem cell‐derived cardiomyocyte; IHD, ischaemic heart disease. Abstract Acute myocardial infarction (MI) remains a major cause of death and disability worldwide.
Melanie Paillard   +29 more
wiley   +1 more source

Molecular profiles of single circulating tumor cells from early breast cancer patients with different lymph node statuses

open access: yesThoracic Cancer, Volume 14, Issue 2, Page 156-167, January 2023., 2023
Circulating tumor cells (CTCs) were clustered into two subtypes significantly correlated with the lymph node metastasis status of early breast cancer patients. Patients with lymph node metastasis have CTCs that express an epithelial/stem‐like pattern of metastasis initiating cells, while those patients without lymph node metastasis have CTCs that have ...
Shuyun Pang   +9 more
wiley   +1 more source

Rho-GTPases subfamily: cellular defectors orchestrating viral infection. [PDF]

open access: yesCell Mol Biol Lett
Zhang B   +5 more
europepmc   +1 more source

Multi‐Omic Analysis Reveals Astrocytic Annexin‐A2 as Critical for Network‐Level Circadian Timekeeping in the Suprachiasmatic Nucleus

open access: yesGlia, EarlyView.
ABSTRACT The mammalian suprachiasmatic nucleus (SCN) orchestrates daily (circadian) rhythms of physiology and behavior by broadcasting timing cues generated autonomously by its mutually reinforcing network of ~10,000 neurons and ~3000 astrocytes. Although astrocytic control of extracellular glutamate and GABA has been implicated in driving circadian ...
Andrew P. Patton   +6 more
wiley   +1 more source

Prion-Dependent Lethality of <i>sup35</i> Missense Mutations Is Caused by Low GTPase Activity of the Mutant eRF3 Protein. [PDF]

open access: yesInt J Mol Sci
Trubitsina NP   +8 more
europepmc   +1 more source

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