Results 141 to 150 of about 6,675 (181)

Challenges in Managing Recurrent Small Bowel Obstruction in a Patient With Concurrent Hepatic Dysfunction and Alcohol Use Disorder: A Multidisciplinary Approach. [PDF]

Cureus
Sevgi K, Enman L, Lucero F, Aran SN.
europepmc   +1 more source

Endocrine-Disrupting Chemicals: A Looming Threat to Current and Future Generations. [PDF]

Int J Mol Sci
Cortes-Ramirez SA, Ho SM, Leung YK.
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Management of Yellow Phosphorus-Induced Acute Liver Failure: A Case Report and Review of Literature. [PDF]

Cureus
R R, Routray M.
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Investigation of the Role of Chemical Analysis in Causality Assessment of Herbal and Dietary Supplement-Induced Liver Injury. [PDF]

Drug Saf
Halegoua-DeMarzio D, Navarro VJ, Davis A, Ahmad J, Avula B, Barnhart H, Barritt AS, Bonkovsky HL, Chen VL, Choi G, Fontana RJ, Ghabril MS, Khan I, Koh C, Odin J, Rockey DC, Rostami H, Serrano J, Sherker AH, Stolz A, Tillmann HL, Vuppalanchi R, Drug-Induced Liver Injury Network.   +22 more
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Microcystin shapes the Microcystis phycosphere through community filtering and by influencing cross-feeding interactions. [PDF]

ISME Commun
Große R, Heuser M, Teikari JE, Ramakrishnan DK, Abdelfattah A, Dittmann E.   +5 more
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Microcystin-LR Drives Early NAFLD Pathogenesis via Hepatic Cholesterol Accumulation: Dysregulation of <i>Ldlr</i> and <i>Abcg1</i> Expression Uncoupled from <i>Srebp2</i>. [PDF]

Toxins (Basel)
Kawahara H, Koto Y, Hitsuda Y, Kurata K, Yoshikiyo K, Hashiguchi A, Maseda H, Okano K, Sugiura N, Shimizu K, Shimizu H.   +10 more
europepmc   +1 more source

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Detection of the cyanobacterial hepatotoxins microcystins

Toxicology and Applied Pharmacology, 2005
Concern regarding the presence of microcystins in drinking water and their possible contamination in food (e.g., salad vegetables, fish, shellfish) has resulted in the need for reliable methods for the detection and accurate quantification of this class of toxins.
Jacqui, McElhiney, Linda A, Lawton
openaire   +2 more sources

Iron as a Hepatotoxin

Digestive Diseases, 1995
Although essential for life, iron in excessive amounts may be toxic. The liver is particularly subject to the toxic effects of iron, since it is the major site of iron storage. Several inherited and acquired human disorders may result in hepatic iron overload, the most common of which are genetic hemochromatosis (GH) and transfusional iron overload. GH
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Papaverine Revisited as a Hepatotoxin

New England Journal of Medicine, 1969
The proliferation of therapeutic agents during the past quarter-century has been accompanied by an increased frequency of drug-induced hepatic disease.1 , 2 Usually, the ability of an agent to produce hepatic injury is recognized within a few years of its introduction into clinical use.
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Hepatotoxin-induced hypertyrosinemia and its toxicological significance

Archives of Toxicology, 2006
A (1)H Nuclear Magnetic Resonance (NMR) spectroscopic investigation of the effects of single doses of four model hepatotoxins on male Sprague-Dawley rats showed that hypertyrosinemia was induced by three of the treatments (ethionine 300 mg/kg, galactosamine hydrochloride 800 mg/kg and isoniazid 400 mg/kg) but not by the fourth (thioacetamide 200 mg/kg).
T Andrew, Clayton, John C, Lindon, Jeremy R, Everett, Claude, Charuel, Gilles, Hanton, Jean-Loic, Le Net, Jean-Pierre, Provost, Jeremy K, Nicholson   +7 more
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