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The incidence of cyanobacterial blooms in freshwaters, including drinking water reservoirs, has increased over the past few decades due to rising nutrient levels. Microcystins are hepatotoxins released from cyanobacteria and have been responsible for the
Linda A Lawton, Peter K J Robertson
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The behaviour of hepatotoxins fromMicrocystis andOscillatoria cyanobacteria in some common water treatment processes was investigated on the laboratory scale in order to obtain data on their potential transfer to drinking water. Two toxins were separated
Heikki Pyysalo, Kaarina Sivonen
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PLANTS AND FUNGI AS HEPATOTOXINS
CHARLES S. DAVIDSON
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Digestive Diseases, 2008
Although essential for life, iron in excessive amounts may be toxic. The liver is particularly subject to the toxic effects of iron, since it is the major site of iron storage. Several inherited and acquired human disorders may result in hepatic iron overload, the most common of which are genetic hemochromatosis (GH) and transfusional iron overload. GH
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Although essential for life, iron in excessive amounts may be toxic. The liver is particularly subject to the toxic effects of iron, since it is the major site of iron storage. Several inherited and acquired human disorders may result in hepatic iron overload, the most common of which are genetic hemochromatosis (GH) and transfusional iron overload. GH
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Investigations into a Hepatotoxin in Lupin Roughage
Research in Veterinary Science, 1970Preliminary investigations into the chemical nature of a hepatotoxic factor which sometimes occurs in dried lupin roughage, particularly in Western Australia, have been carried out. Results indicated it was not an alkaloid. Its properties suggested the presence of phenolic and/or acidic groups.
D S, Petterson, W H, Parr
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Detection of the cyanobacterial hepatotoxins microcystins
Toxicology and Applied Pharmacology, 2005Concern regarding the presence of microcystins in drinking water and their possible contamination in food (e.g., salad vegetables, fish, shellfish) has resulted in the need for reliable methods for the detection and accurate quantification of this class of toxins.
Jacqui, McElhiney, Linda A, Lawton
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Papaverine Revisited as a Hepatotoxin
New England Journal of Medicine, 1969The proliferation of therapeutic agents during the past quarter-century has been accompanied by an increased frequency of drug-induced hepatic disease.1 , 2 Usually, the ability of an agent to produce hepatic injury is recognized within a few years of its introduction into clinical use.
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