Results 91 to 100 of about 111,576 (246)

Genetics of Hereditary Angioedema Revisited

open access: yes, 2016
Contemporary genetic research has provided evidences that angioedema represents a diverse family of disorders related to kinin metabolism, with a much greater genetic complexity than was initially considered.
Germenis A.E., Speletas M.
core   +1 more source

Icatibant, a new bradykinin-receptor antagonist, in hereditary angioedema [PDF]

open access: yes, 2010
BACKGROUND: Hereditary angioedema is characterized by recurrent attacks of angioedema of the skin, larynx, and gastrointestinal tract. Bradykinin is the key mediator of symptoms. Icatibant is a selective bradykinin B2 receptor antagonist.
Wing-Tze Fan   +258 more
core   +1 more source

Hereditary angioedema: how to approach it at the emergency department?

open access: yesEinstein (São Paulo)
Angioedema attacks are common causes of emergency care, and due to the potential for severity, it is important that professionals who work in these services know their causes and management.
Faradiba Sarquis Serpa   +10 more
doaj   +2 more sources

The Pathophysiology of Hereditary Angioedema [PDF]

open access: yesWorld Allergy Organization Journal, 2010
Hereditary angioedema (HAE) causes recurrent episodes of angioedema that may be very severe and are frequently associated with significant morbidity and even mortality. Understanding the pathophysiology of this disease is crucial for proper diagnosis and management of these patients.
openaire   +5 more sources

Sesame Immunotherapy: A Systematic Review and Narrative Synthesis

open access: yesClinical and Translational Allergy, Volume 16, Issue 10, October 2026.
ABSTRACT Sesame allergy is associated with severe allergic reactions, low rates of spontaneous resolution, and significant psychosocial burden. Current management relies on strict avoidance and emergency treatment. Allergen‐specific immunotherapy, particularly oral immunotherapy (OIT), has emerged as a potential disease‐modifying approach, but evidence
Joanna Zielińska   +4 more
wiley   +1 more source

Management of acute attacks of hereditary angioedema: role of ecallantide

open access: yes, 2015
Hannah Duffey,1 Rafael Firszt1,2 1Department of Pediatrics, 2Division of Allergy, Immunology and Rheumatology, University of Utah, Salt Lake City, UT, USA Abstract: Hereditary angioedema (HAE) is characterized as an episodic swelling disorder with ...
Firszt R, Duffey H
core  

Hereditary angioedema in women [PDF]

open access: yesAllergy, Asthma & Clinical Immunology, 2010
Women with hereditary angioedema (HAE) are more likely to be symptomatic that men. Hormonal factors (puberty, contraception, pregnancy,....) play a significant role in the precipitation or worsening of the condition in women. So, combined contraceptive pills are not indicated and progestogen pill must be preferred.
openaire   +3 more sources

Cellular Responses to Tartrazine and Sulfanilic Acid Exposure in BEAS‐2B Cells: Viability, Apoptosis, and DNA Damage

open access: yesJournal of Applied Toxicology, Volume 46, Issue 10, Page 3446-3454, October 2026.
ABSTRACT Tartrazine is a synthetic azo dye widely used in food, pharmaceutical, and cosmetic products, resulting in extensive human exposure, while its toxicity and that of its primary metabolite, sulfanilic acid, remain controversial. Considering the reported association of tartrazine with hypersensitivity and allergic‐like reactions, human bronchial ...
Merve Baysal   +5 more
wiley   +1 more source

Normalization of C1 Inhibitor in a Patient with Hereditary Angioedema [PDF]

open access: yes
Hereditary angioedema is a potentially life-threatening autosomal dominant condition, causing attacks of angioedema due to failure to regulate bradykinin. Nearly all cases of hereditary angioedema are caused by mutations in the gene encoding C1 inhibitor,
Sharif, Khalid   +8 more
core   +1 more source

HEREDITARY ANGIOEDEMA: AN UPDATE. [PDF]

open access: yes, 2015
Objective: Hereditary angioedema is a genetic disease with autosomal dominance inheritance characterized by quantitative and qualitative deficiency of C1 inhibitor, resulting in edema of multiples organs.
Thomaz, José Eduardo   +4 more
core   +1 more source

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