Results 51 to 60 of about 197,953 (330)

The Pathophysiology of Hereditary Angioedema [PDF]

World Allergy Organization Journal, 2010
Hereditary angioedema (HAE) causes recurrent episodes of angioedema that may be very severe and are frequently associated with significant morbidity and even mortality. Understanding the pathophysiology of this disease is crucial for proper diagnosis and management of these patients.
openaire   +6 more sources

Long‐term safety and effectiveness of berotralstat for hereditary angioedema: The open‐label APeX‐S study

Clinical and Translational Allergy, 2021
Background Berotralstat (BCX7353) is an oral, once‐daily inhibitor of plasma kallikrein recently approved for prevention of angioedema attacks in adults and adolescents with hereditary angioedema (HAE).
H. Farkas, M. Stobiecki, J. Peter, T. Kinaciyan, M. Maurer, E. Aygören‐Pürsün, S. Kiani-Alikhan, A. Wu, A. Reshef, A. Bygum, O. Fain, D. Hagin, A. Huissoon, M. Jeseňák, K. Lindsay, V. G. Panovska, U. Steiner, C. Zubrinich, J. Best, M. Cornpropst, D. Dix, S. Dobo, H. Iocca, B. Desai, S. Murray, Enikő Nagy, W. Sheridan   +26 more
semanticscholar   +1 more source

Hereditary Angioedema

Zdorovʹe Rebenka, 2014
Hereditary angioedema — a rare disease caused by a congenital deficiency of C1-inhibitor. Clinical manifestations of the disease — recurrent episodes of angioedema, which manifest in childhood or adolescence, more often localized in the subcutaneous ...
A.P. Volokha
doaj   +1 more source

Classification, diagnosis, and approach to treatment for angioedema: Consensus report from the Hereditary Angioedema International Working Group [PDF]

, 2014
Angioedema is defined as localized and self-limiting edema of the subcutaneous and submucosal tissue, due to a temporary increase in vascular permeability caused by the release of vasoactive mediator(s). When angioedema recurs without significant wheals,
Adam, Agostoni, Agostoni, Agostoni, Alsenz, Alsenz, Bafunno, Bas, Beltrami, Benson, Bentsianov, Binkley, Blais, Blanch, Blohme, Bork, Bork, Bork, Bork, Bork, Bork, Bork, Bork, Bork, Bork, Bork, Bouillet-Claveyrolas, Bowen, Bowen, Branellec, Brasher, Brecy, Bright, Brown, Brown, Burkhart, Bygum, Bygum, Byrd, Byrd, Caballero, Caldwell, Campo, Castelli, Cicardi, Cicardi, Cicardi, Cicardi, Cicardi, Cicardi, Cicardi, Cicardi, Cichon, Cocchio, Colas, Corso, Corte, Craig, Craig, Cugno, Cugno, Cugno, Cugno, Cupido, D'Incan, Dean, Dewald, Dobbels, Donaldson, Donaldson, Donaldson, Du-Thanh, Duan, Farkas, Farkas, Fields, Frank, Gelee, Gelfand, Gompels, Grant, Hassan, Hauptmann, Hauptmann, He, Jackson, Jackson, Jackson, Kalmar, Kalmar, Kaplan, Kiss, Kostis, Kostis, Lam, Levi, Lin, Longhurst, Longhurst, Lopez-Lera, Lopez-Lera, Lumry, Makani, Mandle, Marcos, Martin, Martinez-Saguer, Messerli, Miller, Molinaro, Montinaro, Nielsen, Nussberger, Nussberger, Osler, Pappalardo, Pappalardo, Patel, Pellacani, Quinke, Roche, Roche, Rosen, Rye Rasmussen, Sanchez-Borges, Sanchez-Cano, Sands, Schreiber, Slater, Steinbach, Stray-Pedersen, Tarzi, Tedeschi, Thelwel, Toh, Tosi, Vitrat-Hincky, Wagenaar-Bos, Waytes, Weller, Wilson, Zanichelli, Zanichelli, Ziakas, Zingale, Zingale, Zuberbier, Zuraw, Zuraw, Zuraw, Zuraw, Zuraw   +151 more
core   +1 more source

COVID-19 and hereditary angioedema: Incidence, outcomes, and mechanistic implications.

Allergy and Asthma Proceedings, 2021
Background: Patients with hereditary angioedema (HAE) have been postulated to be at increased risk for coronavirus disease 2019 (COVID-19) infection due to inherent dysregulation of the plasma kallikrein-kinin system.
C. L. Veronez, S. Christiansen, Tukisa D. Smith, M. Riedl, B. Zuraw   +4 more
semanticscholar   +1 more source

International consensus on the diagnosis and management of pediatric patients with hereditary angioedema with C1-Inhibitor deficiency [PDF]

, 2016
BACKGROUND: The consensus documents published to date on hereditary angioedema with C1-inhibitor deficiency (C1-INH-HAE) have focused on adult patients. Many of the previous recommendations have not been adapted to pediatric patients.
Agostoni, Agostoni, Agostoni, Altorjai, Andrew, Assadi, Aygoren-Pursun, Aygoren-Pursun, Bautista-Llacer, Bennett, Berkun, Betschel, Bork, Bork, Bork, Bork, Bork, Bouillet, Bouillet, Bouillet, Bowen, Bowen, Bowen, Bygum, Caballero, Caballero, Caballero, Caballero, Castelli, Chagas, Church, Cicardi, Cicardi, Cicardi, Cicardi, Cicardi, Craig, Craig, Craig, Craig, Craig, Csuka, Cugno, Czaller, Dinkel, Doherty, Ebo, El-Hachem, Fabiani, Farkas, Farkas, Farkas, Farkas, Farkas, Farkas, Farkas, Farkas, Farkas, Foix-L'Helias, Fouche, Gomez-Traseira, Gompels, Grumach, Grumach, Hogasen, Hubiche, Janardhanan, Kalmar, Kaplan, Kelemen, Kim, Kreuz, Kruger, Levy, Li, Lindecken, Longhurst, Lumry, MacGinnitie, Magerl, Martinez-Saguer, Martinez-Saguer, Martinez-Saguer, Martinez-Saguer, McCollough, Nanda, Nielsen, Nurnberger, O'Bier, Ohsawa, Pappalardo, Pappalardo, Pedrosa, Petraroli, Prior, Pritzker, Quintana, Rasmussen, Ren, Riedl, Riedl, Roach, Roche, Sanchez, Sanhueza, Shah, Speletas, Strawczynski, Tuong, Veszeli, Visy, Wahn, Wang, Wasserman, Wilson, Zanichelli, Zanichelli, Zilberberg, Zotter, Zotter, Zuraw, Zuraw, Zuraw   +122 more
core   +3 more sources

Clinical periodontal diagnosis

Periodontology 2000, EarlyView., 2023
Abstract Periodontal diseases include pathological conditions elicited by the presence of bacterial biofilms leading to a host response. In the diagnostic process, clinical signs such as bleeding on probing, development of periodontal pockets and gingival recessions, furcation involvement and presence of radiographic bone loss should be assessed prior ...
Giovanni E. Salvi, Andrea Roccuzzo, Jean‐Claude Imber, Alexandra Stähli, Björn Klinge, Niklaus P. Lang   +5 more
wiley   +1 more source

The diagnosis of hereditary angioedema: family caregivers’ experiences [PDF]

, 2018
The aim of this study was to understand the experiences of family caregivers in the process of diagnosing hereditary angioedema. An interpretive and qualitative research methodology based on Gadamer’s philosophical hermeneutics was carried out.
Cayetano Fernández-Sola, Francisco Sánchez-Hernández, Gadamer H. G., Green J., José Granero-Molina, José Manuel Hernández-Padilla, Laura Helena Antequera-Raynal, María del Mar Jiménez-Lasserrotte   +7 more
core   +1 more source

Current and Prospective Targets of Pharmacologic Treatment of Hereditary Angioedema Types 1 and 2

Clinical reviews in allergy and immunology, 2021
Hereditary angioedema (HAE) is a rare disease that causes episodic attacks of subcutaneous and submucosal edema, which can be painful, incapacitating, and potentially fatal.
Lauré M. Fijen, K. Bork, D. Cohn
semanticscholar   +1 more source

design of the HELP study extension [PDF]

, 2017
Background Hereditary angioedema (HAE) is characterized by recurrent attacks of subcutaneous or submucosal edema. Attacks are unpredictable, debilitating, and have a significant impact on quality of life.
Banerji, Aleena, Baptista, Jovanna, Bernstein, Jonathan A., Busse, Paula J., Cicardi, Marco, Craig, Timothy, Longhurst, Hilary J., Magerl, Markus, Riedl, Marc A., Schranz, Jennifer, Shennak, Mustafa M., Yang, William H.   +11 more
core   +1 more source