Results 131 to 140 of about 40,457 (178)

DNA methylation mediates the multiple sclerosis onset risk associated with HHV-6 DNA positivity. [PDF]

Environ Epigenet
Eisner A, Simpson-Yap S, Park DJ, Morwitch E, Tanner S, Maltby VE, van der Mei I, Lechner-Scott J, Scott RJ, Broadley SA, Lea RA, Stridh P, Olsson T, Jagodic M, Alfredsson L, Taylor BV, Ausimmune Investigators Group , Ponsonby AL.   +17 more
europepmc   +1 more source

Anti-NMDA receptor encephalitis in the setting of inherited chromosomally integrated human herpesvirus 6B (iciHHV-6B): A case report. [PDF]

IDCases
Geiger AM, House WJ, Paez A.
europepmc   +1 more source

Seropositivity against rubella virus envelope protein 1, but not 2, is associated with an increased risk of multiple sclerosis. [PDF]

Clin Transl Immunology
Ingvarsson J, Grut V, Biström M, Berg LP, Butt J, Michels B, Stridh P, Olsson T, Alfredsson L, Kockum I, Waterboer T, Nilsson S, Sundström P.   +12 more
europepmc   +1 more source

Human Endogenous Retroviruses in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome: Emerging Roles in Pathogenesis, Immunity, Biomarkers and Therapeutics. [PDF]

Int J Mol Sci
Perera KD, Oltra E, Carding SR.
europepmc   +1 more source

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Glycoproteins of HHV-6A and HHV-6B

2018
Recently, human herpesvirus 6A and 6B (HHV-6A and HHV-6B) were classified into distinct species. Although these two viruses share many similarities, cell tropism is one of their striking differences, which is partially because of the difference in their entry machinery.
Huamin, Tang, Yasuko, Mori
openaire   +2 more sources

MHC2TA rs4774C and HHV‐6A active replication in multiple sclerosis patients

European Journal of Neurology, 2009
Background and purpose:  In a previous report, a strong gene–environment interaction between human herpesvirus 6A (HHV6A) active replication and MHC2TA rs4774C was demonstrated. The objectives of this study were: (i) to reappraise the association that was found in the previous study; (ii) to evaluate if MS patients with minor allele C and HHV‐6A active
R, Alvarez-Lafuente, A, Martinez, M, Garcia-Montojo, A, Mas, V, De Las Heras, M I, Dominguez-Mozo, C, Maria Del Carmen, M, López-Cavanillas, M, Bartolome, E, Gomez de la Concha, E, Urcelay, R, Arroyo   +11 more
openaire   +2 more sources

HHV-6A infection induces expression of HERV-K18-encoded superantigen

Journal of Clinical Virology, 2009
The human endogenous retrovirus K-18 (HERV-K18) encodes a superantigen that causes deregulation of the immune system. This provirus is transcriptionally silent, but can be induced by Epstein-Barr virus (EBV) infection and IFN-alpha treatment.Since the herpesvirus EBV induces HERV-K18 expression in human B cells, it was of interest to determine if other
Albert K, Tai, Janos, Luka, Dharam, Ablashi, Brigitte T, Huber   +3 more
openaire   +2 more sources

PCR Analysis of Human Telomeric Repeats Present on HHV-6A Viral Strains

Virus Genes, 1997
Human herpesvirus 6 (HHV-6) presents a perfect tandem array of human telomeric repeats (TRS) at both identical direct repeats (DR). Several researchers have reported a different TRS copy number by sequence analysis of HHV-6 DR's cloned fragments so it has been hypothesized that number of TRS is unstable. By PCR we show that the TRS copy number of U1102
MIRANDOLA, Prisco, RAVAIOLI T., CASSAI E.   +2 more
openaire   +3 more sources

AMPK restricts HHV-6A replication by inhibiting glycolysis and mTOR signaling

Virology
AMP-activated protein kinase (AMPK) is a cellular energy sensor regulating metabolic homeostasis. In this study, we investigated the role of AMPK in response to human herpesvirus 6A (HHV-6A) infection. We show that HHV-6A infection significantly downregulates the active phosphorylated state of AMPK in infected T cells.
Xiaodi, Yang, Siyu, Tian, Zhujiang, Min, Emanuela, Garbarino, Jingjing, Ma, Junli, Jia, Huamin, Tang, Lingyun, Li   +7 more
openaire   +2 more sources

HHV-6A, 6B, and 7: persistence in the population: epidemiology and transmission

2007
In common with all human herpesviruses, HHV-6 and HHV-7 establish lifelong infection following initial exposure and seroconversion. True latency as exemplified by HSV-1 and VZV, in which the genome is maintained in a transcriptionally restricted state, has not been conclusively shown for HHV-6 or HHV-7.
Vincent C. Emery, Duncan A. Clark
openaire   +1 more source