Results 41 to 50 of about 307,157 (265)

The homology of the Higman–Thompson groups [PDF]

open access: yesInventiones mathematicae, 2019
49 ...
Szymik, Markus, Wahl, Nathalie
openaire   +5 more sources

Lysyl oxidase (LOX)‐mediated remodeling of the extracellular matrix

open access: yesFEBS Letters, EarlyView.
The lysyl oxidase (LOX) family of enzymes drives extracellular matrix remodeling by oxidizing collagen, elastin, and fibronectin, promoting covalent cross‐link formation. This activity regulates tissue architecture and mechanical properties, while its dysregulation contributes to fibrosis, cardiovascular disease, glaucoma, connective tissue disorders ...
Fernando Rodríguez‐Pascual   +3 more
wiley   +1 more source

(Co)homology of $$\Gamma $$-groups and $$\Gamma $$-homological algebra

open access: yesEuropean Journal of Mathematics, 2022
This is a further investigation of our approach to group actions in homological algebra in the settings of homology of {\Gamma}-simplicial groups, particularly of {\Gamma}-equivariant homology and cohomology of {\Gamma}-groups. This approach could be called {\Gamma}-homological algebra.
openaire   +2 more sources

Homological stability of series of groups [PDF]

open access: yesPacific Journal of Mathematics, 2010
``What aspects of a group are unchanged, or stable, under homology equivalences''? The model theorem in this regard is the 1963 result of J. Stallings that the lower central series is preserved under any integral homological equivalence of groups. Various other theorems of this nature have since appeared.
Cochran, Tim D., Harvey, Shelly
openaire   +3 more sources

KDM7A and KDM1A inhibition suppresses tumour promoting pathways in prostate cancer

open access: yesMolecular Oncology, EarlyView.
Treatment resistance is a major challenge for patients with advanced prostate cancer. This study examined an alternative approach to target the major prostate cancer‐promoting pathway by targeting epigenetic factors, whose levels are higher in tumours.
Jennie N Jeyapalan   +16 more
wiley   +1 more source

Cohomology of Tanabe algebras

open access: yesExtracta Mathematicae
In this paper we study the (co)homology of Tanabe algebras, which are a family of subalgebras of the partition algebras exhibiting a Schur–Weyl duality with certain complex reflection groups.
Andrew Fisher, Daniel Graves
doaj   +1 more source

Transcriptional profiling of circulating extracellular vesicles from prebiopsy prostate cancer patients

open access: yesMolecular Oncology, EarlyView.
RNA profiling of circulating extracellular vesicles (EVs) from blood samples of men undergoing prostate biopsy identifies transcripts associated with clinically significant prostate cancer. Integrative analysis with public tumor datasets links EV‐derived gene signatures to tumor stage and progression‐free survival, highlighting CASP3, XRCC2, and RIT1 ...
Stefan Werner   +14 more
wiley   +1 more source

On the cyclic homology theory of algebras

open access: yesScientific African
In this paper, the (Co)homology theory of pure algebra will be studied. We aim to study the homological theory of cyclic homology and its properties. The triviality property of cohomology groups of algebras will be obtained.
W.M. Mahmoud   +3 more
doaj   +1 more source

Metastasis on pause: How dormant tumor cells stay hidden within the tumor microenvironment and evade immune surveillance

open access: yesMolecular Oncology, EarlyView.
Dormant cancer cells can hide in distant organs for years, evading treatment and the immune system. This review highlights how signals from the surrounding tissue and immune environment keep these cells inactive or trigger their reawakening. Understanding these mechanisms may help develop therapies to eliminate or control dormant cells and prevent ...
Kanishka Tiwary   +1 more
wiley   +1 more source

Loss of IGF‐1R impairs DNA‐PKcs recruitment to chromatin leading to defective end‐joining

open access: yesMolecular Oncology, EarlyView.
IGF‐1R promotes radioresistance by facilitating DNA‐PKcs recruitment to chromatin, enabling non‐homologous end‐joining (NHEJ) repair of double‐strand breaks. Inhibition or loss of IGF‐1R disrupts this recruitment to damage sites, driving compensatory reliance on microhomology‐mediated end‐joining (MMEJ) repair.
Matthew O. Ellis   +3 more
wiley   +1 more source

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