Results 91 to 100 of about 1,464 (122)

The structure of the mammalian bornavirus polymerase complex. [PDF]

open access: yesNat Commun
Carrique L   +4 more
europepmc   +1 more source

Comparison of mutations in human parainfluenza viruses during passage in primary human bronchial/tracheal epithelial air-liquid interface cultures and cell lines. [PDF]

open access: yesMicrobiol Spectr
Sugimoto S   +11 more
europepmc   +1 more source

Mucins and Respiratory Virus Infection. [PDF]

open access: yesAnnu Rev Virol
Mathur S, Corkran M, Scull MA.
europepmc   +1 more source

Half‐Life of Human Parainfluenza Virus Type 3 (hPIV3) Maternal Antibody and Cumulative Proportion of hPIV3 Infection in Young Infants [PDF]

open access: yesJournal of Infectious Diseases, 2001
During a phase 2 trial of parainfluenza virus type 3 (PIV3) vaccine, sequential serum samples were obtained from infants at 2, 6, 7, 12-15, and 13-16 months of age. Paired serum samples obtained at 2 and 6 months of age were used to estimate the biologic half-life of human PIV3 (hPIV3) maternal antibody in young infants.
Min-Shi Lee
exaly   +3 more sources

Recombinant Bovine/Human Parainfluenza Virus Type 3 (B/HPIV3) Expressing the Respiratory Syncytial Virus (RSV) G and F Proteins Can Be Used To Achieve Simultaneous Mucosal Immunization against RSV and HPIV3 [PDF]

open access: yesJournal of Virology, 2001
ABSTRACT Recombinant bovine/human parainfluenza virus type 3 (rB/HPIV3), a recombinant bovine PIV3 (rBPIV3) in which the F and HN genes were replaced with their HPIV3 counterparts, was used to express the major protective antigens of respiratory syncytial virus (RSV) in order to create a bivalent mucosal vaccine against RSV and HPIV3.
Josephine M Mcauliffe, B R Murphy
exaly   +3 more sources

Haemagglutinin-neuraminidase from HPIV3 mediates human NK regulation of T cell proliferation via NKp44 and NKp46 [PDF]

open access: yesJournal of General Virology, 2018
HPIV3 is a respiratory virus causing airway diseases, including pneumonia, croup, and bronchiolitis, during infancy and childhood. Currently there is no effective vaccine or anti-viral therapy for this virus. Studies have suggested that poor T cell proliferation following HPIV3 infection is responsible for impaired immunological memory associated with ...
Sinéad T Loughran   +2 more
exaly   +5 more sources

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