Results 51 to 60 of about 23,066 (306)

Hyperaldosteronism☆

open access: yes, 2007
openAccessArticle: False ; cover date: 2015-01-01 ; pii: B978-0-12-801238-3.05067-4 ; Harvest Date: 2016-01-06 13:08:10 ; issueName: ; Page Range: - ; href scidir: http://www.sciencedirect.com/science/article/pii/B9780128012383050674 ; pubType:
Gail, Galasko, Galasko, Gail
openaire   +2 more sources

Primary hyperaldosteronism presenting as hypokalaemic periodic paralysis

open access: yesSri Lanka Journal of Diabetes Endocrinology and Metabolism, 2019
Hypokalaemic periodic paralysis is episodic painless muscle weakness associated with areflexia (1). Hypertension and hypokalaema are the classic presentation of primary hyperaldoteronism though some patients can be normokalaemic (2).
A. S. Embuldeniya   +3 more
doaj   +1 more source

Familial Hyperaldosteronism Type 3 with a Rapidly Growing Adrenal Tumor: An In Situ Aldosterone Imaging Study

open access: yesCurrent Issues in Molecular Biology, 2021
Primary aldosteronism is most often caused by aldosterone-producing adenoma (APA) and bi-lateral adrenal hyperplasia. Most APAs are caused by somatic mutations of various ion channels and pumps, the most common being the inward-rectifying potassium ...
Nae Takizawa   +12 more
doaj   +1 more source

Vascular dysfunction and fibrosis in stroke-prone spontaneously hypertensive rats: the aldosterone-mineralocorticoid receptor-Nox1 Axis [PDF]

open access: yes, 2017
Aims: We questioned whether aldosterone and oxidative stress play a role in vascular damage in severe hypertension and investigated the role of Nox1 in this process.
Ceravolo, Graziela   +7 more
core   +1 more source

The prevalence of resistant arterial hypertension and secondary causes in a cohort of hypertensive patients: a single center experience [PDF]

open access: yes, 2017
The prevalence of resistant hypertension (RHT) still remains unknown. Aim of the study was to investigate in a large cohort of hypertensive patients the prevalence of RHT, and to identify in these patients the secondary forms of arterial hypertension (SH)
Concistre, Antonio   +13 more
core   +2 more sources

Primary Hyperaldosteronism and Renal Medullary Nephrocalcinosis: A Controversial Association

open access: yesOman Medical Journal, 2021
Primary hyperaldosteronism (PA) is a common disease with a prevalence of 5–10% in unselected patients with hypertension. Medullary nephrocalcinosis is a radiological diagnosis and refers to diffuse calcification in the renal parenchyma.
Raiz Ahmad Misgar   +3 more
doaj   +1 more source

A case report of hyponatremia after surgery for Conn's adenoma [PDF]

open access: yes, 2017
Primary aldosteronism (PA), also known as Conn's syndrome, is a frequent cause of secondary hypertension. If PA is due to a documented unilateral adrenal adenoma, adrenalectomy is the treatment of choice.
Bardelli, Moreno   +6 more
core   +1 more source

’Malignant’ hypertension from hyperaldosteronism: a case report

open access: yesThe Pan African Medical Journal, 2018
Adrenocortical carcinomas (ACC) are rare with an incidence of 0.7-2 per million population per year and account for only 0.05%-2% of all malignant tumors. While majority of the functional ACC present as Cushing syndrome, recurrent hyperaldosteronism from
Krishna Mohan Baradhi   +2 more
doaj   +1 more source

Junctional Bradycardia as Early Sign of Digoxin Toxicity in a Premature Infant with Congestive Heart Failure due to a Left to Right Shunt

open access: yesAmerican Journal of Perinatology Reports, 2016
Introduction Congestive heart failure due to left to right cardiac shunt is usually managed medically with diuretics, angiotensin converting enzyme inhibitors, and, in some cases, with the addition of digoxin.
Soham Dasgupta   +2 more
doaj   +1 more source

Pathogenesis of hypertension in a mouse model for human CLCN2 related hyperaldosteronism

open access: yesNature Communications, 2019
Human primary aldosteronism (PA) can be caused by mutations in several ion channel genes but mouse models replicating this condition are lacking. We now show that almost all known PA-associated CLCN2 mutations markedly increase ClC-2 chloride currents ...
Corinna Göppner   +8 more
semanticscholar   +1 more source

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