Results 171 to 180 of about 20,403 (220)
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Journal of Experimental Biology, 1967
ABSTRACT The current view of the control feeding in the blowfly Phormia regina Meigen holds that ingestion is initiated by stimulation of oral taste receptors and ultimately terminated by signals from the foregut (Dethier & Bodenstein, 1958).
V. G. Dethier, A. Gelperin
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ABSTRACT The current view of the control feeding in the blowfly Phormia regina Meigen holds that ingestion is initiated by stimulation of oral taste receptors and ultimately terminated by signals from the foregut (Dethier & Bodenstein, 1958).
V. G. Dethier, A. Gelperin
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Prolactin and the hyperphagia of lactation
Physiology & Behavior, 2007The nutritional needs of developing young place a large energetic demand on lactating females. In this paper some of the mechanisms through which lactating rats meet these demands are described. Emphasis is placed on the mechanisms that drive increased food intake in suckled rats that are independent of milk delivery and negative energy balance ...
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Growth, thermogenesis, and hyperphagia
The American Journal of Clinical Nutrition, 1990Resting metabolic rate is demonstrated to be a function of fat-free mass and a growth variable related to food-energy-input imbalance rate. By use of obligatory energy expenditure terms, the two-reservoir energy model applied to hyperphagia shows that growth of the fat-free mass is rapid whereas that of the fat store is slow and that the growth of both
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Metabolic effects of hypothalamic hyperphagia
Metabolism, 1981In order to test the hypothesis that the enhanced gluconeogenesis of hypothalamic obesity remains responsive to changed in food intake, we have measured gluconeogenesis in two modes of hypothalamic obesity under both hyperphagic and normophagic conditions.
R G, MacKenzie +3 more
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Hyperphagia: A Necessary Precondition to Obesity?
Appetite, 1985Weight-gain is generally attributed to a caloric imbalance resulting from hyperphagia. However, this attribution is often made without observing caloric intakes during the initial accumulation of fat stores. Instead, this conclusion is drawn because many obese organisms overeat, and overconsumption is sufficient to cause weight-gain.
J M, Slattery, R M, Potter
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Hyperphagia induced by pylorectomy in sheep
Physiology & Behavior, 1989In the hay-fed sheep, gastric (abomasal) contents are delivered continuously into the duodenal bulb at a mean flow rate of 210 ml per hour. The pylorus could behave as the site of origin of a feedback satiety signal by acting as a distal stomach sphincter limiting the abomasal emptying rate.
Malbert, Charles-Henri, Ruckebusch, Y.
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Hyperphagia in Ruminants Induced by a Depressant
Science, 1966Attempts at causing ventromedial hyperphagia in ruminants have been hitherto unsuccessful. In our experiments perfusion of the ventriculocisternal system with pentobarbital caused marked hyperphagia. This suggests that the ventromedial hypothalamic area is functioning in ruminants, probably as in monogastric animals, by inhibiting the lateral area.
C A, Baile, J, Mayer
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JAMA, 1981
Progressive left hemiparesis followed by face and trunk cutaneous vasodilation and hyperphagia developed in a 28-year-old man. He began eating five to six meals a day and gained 16 kg in 60 days. Computed tomography disclosed a neoplastic lesion involving the midline via the hypothalamus and reaching the contralateral lenticular nucleus.
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Progressive left hemiparesis followed by face and trunk cutaneous vasodilation and hyperphagia developed in a 28-year-old man. He began eating five to six meals a day and gained 16 kg in 60 days. Computed tomography disclosed a neoplastic lesion involving the midline via the hypothalamus and reaching the contralateral lenticular nucleus.
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Science, 1975
Major differences were found between classical hypothalamic hyperphagia in rats and the recently discovered hyperphagia syndrome resulting from destruction of the ventral noradrenergic bundle. Traditional medial hypothalamic lesions produced no detectable loss of norepinephrine, and the rats overate both in the daytime and at night, whereas destruction
J E, Ahlskog, P K, Randall, B G, Hoebel
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Major differences were found between classical hypothalamic hyperphagia in rats and the recently discovered hyperphagia syndrome resulting from destruction of the ventral noradrenergic bundle. Traditional medial hypothalamic lesions produced no detectable loss of norepinephrine, and the rats overate both in the daytime and at night, whereas destruction
J E, Ahlskog, P K, Randall, B G, Hoebel
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Behavioral and Neural Biology, 1979
Bilateral electrolytic lesions were made in female Wistar rats to compare the characteristics of the obesity produced by lesioning the mesencephalon (MES) with those produced by lesioning the ventromedial hypothalamus (VMH). The hyperphagia following VMH lesions was immediate but it developed more slowly in the rats with MES lesions and in several rats
B M, Box, R, Bascom, G J, Mogenson
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Bilateral electrolytic lesions were made in female Wistar rats to compare the characteristics of the obesity produced by lesioning the mesencephalon (MES) with those produced by lesioning the ventromedial hypothalamus (VMH). The hyperphagia following VMH lesions was immediate but it developed more slowly in the rats with MES lesions and in several rats
B M, Box, R, Bascom, G J, Mogenson
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