Results 41 to 50 of about 749,882 (394)

Soluble ST2 levels and left ventricular structure and function in patients with metabolic syndrome [PDF]

open access: yes, 2016
Background: A biomarker that is of great interest in relation to adverse cardiovascular events is soluble ST2 (sST2), a member of the interleukin family.
Beunza, Maite   +14 more
core   +1 more source

Ventricular androgenic-anabolic steroid-related remodeling: an immunohistochemical study [PDF]

open access: yes, 2017
Background: Several fatal cases of bodybuilders, following a myocardial infarction after long exposure to androgenicanabolic steroids (AAS), are reported. In recent years, evidence has emerged of cases of heart failure related to AAS consumption, with no
Cecchi, Rossana   +7 more
core   +1 more source

Cardiac Overexpression of S100A6 Attenuates Cardiomyocyte Apoptosis and Reduces Infarct Size After Myocardial Ischemia‐Reperfusion

open access: yesJournal of the American Heart Association: Cardiovascular and Cerebrovascular Disease, 2017
BackgroundCardiomyocyte‐specific transgenic mice overexpressing S100A6, a member of the family of EF‐hand calcium‐binding proteins, develop less cardiac hypertrophy, interstitial fibrosis, and myocyte apoptosis after permanent coronary ligation, findings
Azadeh Mofid   +13 more
doaj   +1 more source

Risk factors for obstructive sleep apnea syndrome in children: state of the art [PDF]

open access: yes, 2019
The obstructive sleep apnea syndrome (OSAS) represents only part of a large group of pathologies of variable entity called respiratory sleep disorders (RSD) which include simple snoring and increased upper airway resistance syndrome (UARS).
Bellini, Chiara   +16 more
core   +2 more sources

Hypertrophy of the Turbinate. [PDF]

open access: yesJAMA: The Journal of the American Medical Association, 1907
Brookings, S. D., March 4, 1907. To the Editor: —After reading with interest the article of Dr. Kuyk inThe Journalof March 2, in which he describes his method of dealing with hypertrophy of the inferior turbinate, I am prompted to describe a method which has proved successful in my hands. The observations of Dr.
openaire   +2 more sources

Heart-Healthy Hypertrophy [PDF]

open access: yesCell Metabolism, 2011
Exercise induces growth of heart muscle cells and heart size. A new report in Cell (Boström et al., 2010) suggests that mice also respond to exercise with increased cardiac myocyte proliferation, and the molecular regulators of this pathway are linked to maladaptive and pathologic responses to cardiac stresses such as pressure overload.
Jonathan A. Epstein, Chinmay M. Trivedi
openaire   +3 more sources

A review of the molecular mechanisms underlying the development and progression of cardiac remodeling [PDF]

open access: yes, 2017
Pathological molecular mechanisms involved in myocardial remodeling contribute to alter the existing structure of the heart, leading to cardiac dysfunction.
et al,, Yee, Derek
core   +7 more sources

Emerging Roles of Neuronal Ca2+ Sensor-1 in Cardiac and Neuronal Tissues: A Mini Review

open access: yesFrontiers in Molecular Neuroscience, 2019
The EF-hand calcium (Ca2+)-binding protein, neuronal Ca2+ sensor-1 (NCS-1/frequenin), is predominantly expressed in neuronal tissues and plays a crucial role in neuronal functions, including synaptic transmission and plasticity.
Tomoe Y. Nakamura   +4 more
doaj   +1 more source

Mechanisms of exercise-induced improvements in the contractile apparatus of the mammalian myocardium [PDF]

open access: yes, 2010
One of the main outcomes of aerobic endurance exercise training is the improved maximal oxygen uptake, and this is pivotal to the improved work capacity that follows the exercise training.
Adams   +98 more
core   +1 more source

The N6-Methyladenosine mRNA Methylase METTL3 Controls Cardiac Homeostasis and Hypertrophy

open access: yesCirculation, 2018
Background: N6-Methyladenosine (m6A) methylation is the most prevalent internal posttranscriptional modification on mammalian mRNA. The role of m6A mRNA methylation in the heart is not known. Methods: To determine the role of m6A methylation in the heart,
L. Dorn   +8 more
semanticscholar   +1 more source

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