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Hepatitis A vaccination during ventricular assist device support in heart transplant candidates: immunogenicity and safety in a single-center cohort. [PDF]
Arima M +7 more
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Comparative Epidemiological and Metabolic Profiling in Alcoholic Versus Metabolic Dysfunction-Associated Steatotic Liver Disease: A Western India Cross-Sectional Analysis. [PDF]
Dhanavade SS, Kshirsagar MS, Vani AC.
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Inclusion Body Hepatitis as a Primary Disease in Broilers in Saskatchewan, Canada
Avian Diseases, 2006In recent years inclusion body hepatitis (IBH) has emerged as an economically important disease in Western Canada. Historically, infections with infectious bursal disease virus (IBDV) and chicken anemia virus (CAV) have been known to suppress the immune system of broilers and make them more susceptible to a secondary disease such as IBH.
Davor Ojkic +2 more
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Inclusion-Body Hepatitis in Broiler Chickens
Avian Diseases, 1972SUMMARY Eighty-six outbreaks of inclusion-body hepatitis were identified in broiler chickens in Ontario. The disease occurred mainly in birds 5 weeks old, and mortality (ranging from 0.1 to 7%) was most common in the spring. Grossly, the common lesions were stellate or punctiform hemorrhagic areas in extremely fatty livers, enlarged pale kidneys ...
J R, Pettit, H C, Carlson
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INCLUSION BODY HEPATITIS IN A PRAIRIE FALCON
Journal of Wildlife Diseases, 1971Intranuclear inclusion bodies, typical of the developmental stages of herpesvirus inclusions, were observed in cells of the liver, spleen, bone marrow, and small intestine from a prairie falcon (Falco mexicanus). Inoculations of a suspension of liver tissue produced lesions on the chorioallantoic membranes of 12-day old chicken embryos.
F P, Ward, D G, Fairchild, J V, Vuicich
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Psittacine inclusion body hepatitis in an aviary
Journal of the American Veterinary Medical Association, 1985Psittacine inclusion body hepatitis (also known as Pacheco's parrot disease) was believed to be responsible for fatal necrotizing hepatitis and splenitis in a variety of psittacine birds from a private aviary. Splenic cells and degenerative hepatocytes around the outer zone of necrotic areas had margination of nuclear material and large intranuclear ...
M, Cartwright +2 more
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Avian Pathology, 1977
The fine structure of intranuclear inclusion bodies in the hepatic cells of chickens naturally affected with inclusion body hepatitis was studied comparing them with the inclusions seen with light microscopy. Ultra-structurally the inclusions were classified into two types depending on whether they contained virus particles or not.
C, Itakura, S, Matsushita, M, Goto
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The fine structure of intranuclear inclusion bodies in the hepatic cells of chickens naturally affected with inclusion body hepatitis was studied comparing them with the inclusions seen with light microscopy. Ultra-structurally the inclusions were classified into two types depending on whether they contained virus particles or not.
C, Itakura, S, Matsushita, M, Goto
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Inclusion-Body Hepatitis in Day-Old Turkeys
Avian Diseases, 1988Inclusion-body hepatitis was diagnosed in 1-day-old turkeys experiencing above-average mortality. At necropsy, turkeys appeared anemic and had pale yellow livers. Histopathologic examination of affected livers revealed diffuse hepatic degeneration and multifocal necrosis, with approximately 70% of the hepatocytes containing large, basophilic ...
J S, Guy, J L, Schaeffer, H J, Barnes
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Antigenic Characterization of the Inclusion Body Hepatitis Virus
American Journal of Veterinary Research, 1975SUMMARY Proof that inclusion body hepatitis of chickens (ibhc) is caused by a virus (ibhv) was established in 1973. Characterization of ibhv has been conducted by employing the standard criteria for virus classification. The results have indicated that ibhv is a member of the avian adenovirus group.
A M, Fadly, R W, Winterfield
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A Primary Epidemic of Inclusion Body Hepatitis in Broilers
Avian Diseases, 1989Inclusion body hepatitis (IBH) was diagnosed in 15 broiler flocks supplied by one breeder in the South Island of New Zealand. The affected flocks suffered mortality up to 30%. Malaise and slightly increased mortality were noticed by growers from about day 12 post-hatch; mortality peaked in the fourth week, and, in most flocks, declined to normally ...
N H, Christensen, M, Saifuddin
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