Results 111 to 120 of about 22,370 (168)
A Systematic Review and Meta-Analysis of Malnutrition and Metabolic Failure in High-Potency Incretin Therapy. [PDF]
Ampofo E +3 more
europepmc +1 more source
Correction to: Should Incretin Agonist-Based Drugs be Considered for First Line Antihypertensive Therapy? [PDF]
Kylies D, Dreher L, Wenzel UO.
europepmc +1 more source
AI-enabled chest x-ray potentially detects subclinical diastolic dysfunction in diabesity and monitors its functional responses to GLP-1 or GLP-1/GIP receptor agonists: initial exploration and proof-of-concept. [PDF]
White RD, Demirer M, Erdal BS, Lin G.
europepmc +1 more source
Do Fasting GLP-1 and GIP Levels Predict the Initial Pharmacological Response to Semaglutide and Tirzepatide? [PDF]
La Vignera S +2 more
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Journal of Intensive Care Medicine, 2015
Glucose-dependent insulinotropic polypeptide (GIP) and glucagon-like peptide-1 (GLP-1) are the known incretin hormones in humans, released predominantly from the enteroendocrine K and L cells within the gut. Their secretion is regulated by a complex of integrated mechanisms involving direct contact for the activation of different chemo-sensors on the ...
Tongzhi, Wu +2 more
openaire +4 more sources
Glucose-dependent insulinotropic polypeptide (GIP) and glucagon-like peptide-1 (GLP-1) are the known incretin hormones in humans, released predominantly from the enteroendocrine K and L cells within the gut. Their secretion is regulated by a complex of integrated mechanisms involving direct contact for the activation of different chemo-sensors on the ...
Tongzhi, Wu +2 more
openaire +4 more sources
The role of incretins and incretin-based drugs in autoimmune diseases
International Immunopharmacology, 2021Incretin hormones, including glucagon-like peptide (GLP)-1, GLP-2 and glucose-dependent insulinotropic polypeptide (GIP), are gastrointestinal peptides secreted from enteroendocrine cells. These hormones play significant roles in many physiological processes via binding to G-protein coupled receptors (GPCRs) on different organs and tissues; one of them
Shabnam Radbakhsh +3 more
openaire +2 more sources
Mayo Clinic Proceedings, 1988
A greater plasma concentration of insulin after isoglycemic enteral than after parenteral administration of glucose is called the incretin effect. The primary mediator of this effect, gastric inhibitory polypeptide, may not account for the complete manifestation of this phenomenon.
L T, Shuster +4 more
openaire +2 more sources
A greater plasma concentration of insulin after isoglycemic enteral than after parenteral administration of glucose is called the incretin effect. The primary mediator of this effect, gastric inhibitory polypeptide, may not account for the complete manifestation of this phenomenon.
L T, Shuster +4 more
openaire +2 more sources
Medical Clinics of North America, 2015
Incretin-based therapies are steadily gaining clinical popularity, with many more products in the developmental pipeline. Current treatment recommendations incorporate GLP-1 RAs and DPP-4 inhibitors as important agents for consideration in the treatment of T2DM owing to their low hypoglycemia risk, ability to address postprandial hyperglycemia (DPP-4 ...
exaly +3 more sources
Incretin-based therapies are steadily gaining clinical popularity, with many more products in the developmental pipeline. Current treatment recommendations incorporate GLP-1 RAs and DPP-4 inhibitors as important agents for consideration in the treatment of T2DM owing to their low hypoglycemia risk, ability to address postprandial hyperglycemia (DPP-4 ...
exaly +3 more sources
Scandinavian Journal of Clinical and Laboratory Investigation, 2001
Incretin hormones are insulinotropic hormones from the intestinal mucosa, which after being released in response to ingestion of a meal, enhance insulin secretion in excess of that elicited by the absorbed nutrients (glucose. amino acids etc) themselves.
Holst, J.J., Orskov, C.
openaire +2 more sources
Incretin hormones are insulinotropic hormones from the intestinal mucosa, which after being released in response to ingestion of a meal, enhance insulin secretion in excess of that elicited by the absorbed nutrients (glucose. amino acids etc) themselves.
Holst, J.J., Orskov, C.
openaire +2 more sources

