Results 181 to 190 of about 2,081,675 (219)
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Current Opinion in Clinical Nutrition and Metabolic Care, 2013
Inflamm-ageing, defined as the chronic low-grade inflammation typical of ageing, seems to be the common biological factor responsible for the decline and the onset of disease in the elderly. The major age-related diseases share a common inflammatory pathogenesis, giving rise to the so-called 'diseasome of inflamm-ageing'.
E. Cevenini +2 more
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Inflamm-ageing, defined as the chronic low-grade inflammation typical of ageing, seems to be the common biological factor responsible for the decline and the onset of disease in the elderly. The major age-related diseases share a common inflammatory pathogenesis, giving rise to the so-called 'diseasome of inflamm-ageing'.
E. Cevenini +2 more
openaire +3 more sources
Current Pharmaceutical Design, 2010
The inflammatory process in arthropods includes primarily the recruitment of circulating hemocytes to wounds or sites of microbial infections. Melanization, capsule formation and clotting reactions will finally result in the sealing of wounds. In this review we will focus on recent research about hemolymph clotting and melanization reactions, and the ...
Pikul, Jiravanichpaisal +2 more
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The inflammatory process in arthropods includes primarily the recruitment of circulating hemocytes to wounds or sites of microbial infections. Melanization, capsule formation and clotting reactions will finally result in the sealing of wounds. In this review we will focus on recent research about hemolymph clotting and melanization reactions, and the ...
Pikul, Jiravanichpaisal +2 more
openaire +2 more sources
Annual Review of Biochemistry, 2016
Necroptosis is a regulated form of necrosis, with the dying cell rupturing and releasing intracellular components that can trigger an innate immune response. Toll-like receptor 3 and 4 agonists, tumor necrosis factor, certain viral infections, or the T cell receptor can trigger necroptosis if the activity of the protease caspase-8 is compromised ...
Kim, Newton, Gerard, Manning
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Necroptosis is a regulated form of necrosis, with the dying cell rupturing and releasing intracellular components that can trigger an innate immune response. Toll-like receptor 3 and 4 agonists, tumor necrosis factor, certain viral infections, or the T cell receptor can trigger necroptosis if the activity of the protease caspase-8 is compromised ...
Kim, Newton, Gerard, Manning
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Journal of Endotoxin Research, 2001
Severe infection induces both activation of the coagulation system and multiple other inflammatory mediator cascades. This concise review summarizes the current knowledge of mechanisms that are considered to contribute to the procoagulant response to sepsis.
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Severe infection induces both activation of the coagulation system and multiple other inflammatory mediator cascades. This concise review summarizes the current knowledge of mechanisms that are considered to contribute to the procoagulant response to sepsis.
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Critical Care Medicine, 2010
In the pathogenesis of sepsis, inflammation and coagulation play a pivotal role. Increasing evidence points to an extensive cross-talk between these two systems, whereby inflammation leads to activation of coagulation, and coagulation also considerably affects inflammatory activity.
Marcel, Levi, Tom, van der Poll
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In the pathogenesis of sepsis, inflammation and coagulation play a pivotal role. Increasing evidence points to an extensive cross-talk between these two systems, whereby inflammation leads to activation of coagulation, and coagulation also considerably affects inflammatory activity.
Marcel, Levi, Tom, van der Poll
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Brain, Behavior, and Immunity
Comments on an article by Silvia Castany et al. Inflammatory processes underlie or contribute to not only a sur prisingly wide array of serious physical and mental disorders, but also play important roles in non-disease-related adaptive neural and biobehavioral processes.
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Comments on an article by Silvia Castany et al. Inflammatory processes underlie or contribute to not only a sur prisingly wide array of serious physical and mental disorders, but also play important roles in non-disease-related adaptive neural and biobehavioral processes.
openaire +3 more sources
Nature Immunology, 2005
This series of reviews addresses mechanisms whereby inflammation can be reduced, turned off or perhaps prevented, both in the context of natural dampening processes as well as possible approaches to anti-inflammatory therapy. Underlying the central issue is the implication that inflammation is indeed something inherently bad that needs to be dampened ...
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This series of reviews addresses mechanisms whereby inflammation can be reduced, turned off or perhaps prevented, both in the context of natural dampening processes as well as possible approaches to anti-inflammatory therapy. Underlying the central issue is the implication that inflammation is indeed something inherently bad that needs to be dampened ...
openaire +2 more sources
Proceedings of the American Thoracic Society, 2005
Abstract Acute and chronic lung diseases are almost invariably associated with some degree of inflammation. Cells that evolved as an effective mechanism to counter infection and heal lung tissue may, in some circumstances, themselves be partially responsible for the pathogenesis of chronic lung disease that leads to irreversible lung ...
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Abstract Acute and chronic lung diseases are almost invariably associated with some degree of inflammation. Cells that evolved as an effective mechanism to counter infection and heal lung tissue may, in some circumstances, themselves be partially responsible for the pathogenesis of chronic lung disease that leads to irreversible lung ...
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Inflammation in Atherosclerosis
2017Atherosclerosis is a disease of the cardiovascular system and its clinical manifestations account for a considerable portion of deaths worldwide. While pathogenesis is initiated by defects in the arterial vasculature and in lipoprotein metabolism, the accompanying sterile inflammation is a fundamental aspect of disease progression.
Lauterbach, Mario A. R. +2 more
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