Results 61 to 70 of about 1,259,702 (421)

Alterations of the CARD15/NOD2 gene and the impact on management and treatment of Crohn's disease patients [PDF]

open access: yes, 2003
The recent identification of the CARD15/NOD2 gene as a susceptibility locus for Crohn's disease represents an important step towards the delineation of the immuno-pathogenesis of inflammatory bowel disease.
Folwaczny, Christian   +3 more
core   +1 more source

Pouchitis in inflammatory bowel disease: a review of diagnosis, prognosis, and treatment [PDF]

open access: yesIntestinal Research, 2021
Patients with inflammatory bowel disease (IBD) occasionally need a restorative proctocolectomy with ileal pouch-anal anastomosis (IPAA) because of medically refractory colitis or dysplasia/cancer.
Shintaro Akiyama   +2 more
doaj   +1 more source

Toward an integrated clinical, molecular and serological classification of inflammatory bowel disease: report of a Working Party of the 2005 Montreal World Congress of Gastroenterology.

open access: yesCanadian journal of gastroenterology = Journal canadien de gastroenterologie, 2005
The discovery of a series of genetic and serological markers associated with disease susceptibility and phenotype in inflammatory bowel disease has led to the prospect of an integrated classification system involving clinical, serological and genetic ...
M. Silverberg   +21 more
semanticscholar   +1 more source

Inflammatory bowel disease biomarkers

open access: yesMedicinal research reviews (Print), 2022
Inflammatory bowel disease (IBD) is characterized as chronic inflammation in the gastrointestinal tract, which includes two main subtypes, Crohn's disease and ulcerative colitis.
Dandan Liu   +4 more
semanticscholar   +1 more source

Using gene expression data to identify certain gastro-intestinal diseases [PDF]

open access: yes, 2012
BACKGROUND: Inflammatory bowel diseases, ulcerative colitis and Crohn’s disease are considered to be of autoimmune origin, but the etiology of irritable bowel syndrome remains elusive.
David A Schwartz   +8 more
core   +1 more source

GPR65 promotes intestinal mucosal Th1 and Th17 cell differentiation and gut inflammation through downregulating NUAK2

open access: yesClinical and Translational Medicine, 2022
G protein‐coupled receptor 65 (GPR65), a susceptibility gene for inflammatory bowel diseases (IBD), has been identified to promote Th17 cell pathogenicity and induce T cell apoptosis.
Ritian Lin   +9 more
doaj   +1 more source

Candidate genes colocalized to linkage regions in inflammatory bowel disease [PDF]

open access: yes, 2002
Background and Aims: The genes encoding for tumor necrosis factor-alpha (TNF-alpha), epidermal growth factor receptor (EGFR) and the vitamin D receptor (VDR) are colocalized to inflammatory bowel disease-associated linkage regions on chromosomes 6, 7 and
Borchers, R.   +4 more
core   +1 more source

Enterogenous Microbiotic Markers in the Differential Diagnosis of Crohn’s Disease and Intestinal Tuberculosis

open access: yesFrontiers in Immunology, 2022
Crohn’s disease (CD) is a chronic intestinal disorder characterized by refractory gastrointestinal ulcerations. Intestinal tuberculosis (ITB) is one common intestinal disease in east Asia.
Mingshan Jiang   +29 more
doaj   +1 more source

Cytokines in inflammatory bowel disease [PDF]

open access: yesMediators of Inflammation, 1997
Over the past decade, much has been learned regarding the role of various cytokines in the pathogenesis of inflammatory bowel disease. Several cytokine ‘knockout’ models in mice have been shown to develop colitis, while alterations in the production of various cytokines has been documented in human Crohn′s disease and ulcerative colitis.
Beck, P. L., Wallace, J. L.
openaire   +5 more sources

Prevalence of inflammatory bowel disease among coeliac disease patients in a Hungarian coeliac centre [PDF]

open access: yes, 2015
BACKGROUND: Celiac disease, Crohn disease and ulcerative colitis are inflammatory disorders of the gastrointestinal tract with some common genetic, immunological and environmental factors involved in their pathogenesis.
A Culliford   +48 more
core   +1 more source

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