Results 31 to 40 of about 1,122,542 (370)

Regulation of Type 2 Immunity in Myocardial Infarction

open access: yesFrontiers in Immunology, 2019
Type 2 immunity participates in the pathogeneses of helminth infection and allergic diseases. Emerging evidence indicates that the components of type 2 immunity are also involved in maintaining metabolic hemostasis and facilitating the healing process ...
Jun-Yan Xu   +3 more
doaj   +1 more source

Interleukin-2 drives cortisol secretion in an age-, dose-, and body composition-dependent way

open access: yesEndocrine Connections, 2020
Background: Interleukin-2 (IL-2), one of the proinflammatory cytokines, is used in the treatment of certain malignancies. In some studies, transient i ncreases in cortisol and ACTH secretion occurred.
Ferdinand Roelfsema   +4 more
doaj   +1 more source

C-reactive protein, interleukin 6, and risk of developing type 2 diabetes mellitus.

open access: yesJournal of the American Medical Association (JAMA), 2001
CONTEXT Inflammation is hypothesized to play a role in development of type 2 diabetes mellitus (DM); however, clinical data addressing this issue are limited.
A. Pradhan   +4 more
semanticscholar   +1 more source

Predictability of IL-28B-polymorphism on protease-inhibitor-based triple-therapy in chronic HCV-genotype-1 patients: A meta-analysis [PDF]

open access: yesWorld Journal of Hepatology, 6(10):759-765, 2014, 2014
AIM: To investigate the predictability of interleukin-28B single nucleotide polymorphism rs12979860 with respect to sustained virological response (SVR) in chronically hepatitis C virus (HCV) genotype-1 patients treated with a protease-inhibitor and pegylated interferon-$\alpha$ (Peg-INF-$\alpha$) based triple-therapy.
arxiv   +1 more source

Keratin-Butyrate Scaffolds Promote Skin Wound Healing in Diabetic Rats Through Down-Regulation of IL-1β and Up-Regulation of Keratins 16 and 17

open access: yesJournal of Natural Fibers, 2023
Impaired wound healing particularly in diabetics creates a significant healthcare burden. The study aimed to evaluate the effect of keratin-butyrate fibers (FKDP +0.1%NaBu) in a full-thickness skin wound model in 30 diabetic rats.
Marek Konop   +14 more
doaj   +1 more source

Divergent dynamics of inflammatory mediators and multiplex PCRs during airway infection in cystic fibrosis patients and healthy controls: Serial upper airway sampling by nasal lavage

open access: yesFrontiers in Immunology, 2022
BackgroundIn cystic fibrosis (CF), acute respiratory exacerbations critically enhance pulmonary destruction. Since these mainly occur outside regular appointments, they remain unexplored.
Nina Erdmann   +11 more
doaj   +1 more source

Interleukin 10(IL-10) inhibits cytokine synthesis by human monocytes: an autoregulatory role of IL-10 produced by monocytes

open access: yesJournal of Experimental Medicine, 1991
In the present study we demonstrate that human monocytes activated by lipopolysaccharides (LPS) were able to produce high levels of interleukin 10 (IL-10), previously designated cytokine synthesis inhibitory factor (CSIF), in a dose dependent fashion. IL-
R. de Waal Malefyt   +4 more
semanticscholar   +1 more source

Anti-inflammatory effects of shikonin in human periodontal ligament cells

open access: yesPharmaceutical Biology, 2018
Context: Shikonin (SHI), an active component extracted from Radix Arnebiae, has been reported to possess anti-inflammatory properties in various cells.
Chen Fan, Xufang Zhang, Zee Upton
doaj   +1 more source

Bullous pemphigoid—What do we know about the most recent therapies?

open access: yesFrontiers in Medicine, 2022
IntroductionBullous pemphigoid (BP) is the most common subtype of autoimmune blistering diseases that primarily affects the elderly and is classically defined by the presence of IgG and/or complement C3 against the BP180 and BP230 hemidesmosome proteins.
Faith A. P. Zeng   +3 more
doaj   +1 more source

A qualitative analysis of a A$β$-monomer model with inflammation processes for Alzheimer's disease [PDF]

open access: yesarXiv, 2022
We introduce and study a new model for the progression of Alzheimer's disease incorporating the interactions of A$\beta$-monomers, oligomers, microglial cells and interleukins with neurons through different mechanisms such as protein polymerization, inflammation processes and neural stress reactions.
arxiv  

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