Results 31 to 40 of about 2,814,914 (235)

Patterns and Reasons for Junk Food Consumption among Young Adults: A Mixed Methods Study from a Medical College in South India

open access: yesArchives of Medicine and Health Sciences
Background and Aim: Food item that has little nutritive value, high fat, and sugar content with little to no fiber content is labeled as junk food. The objective of the study is to assess the patterns of consumption of junk food among young adults and to
Poonam Ramesh Naik   +2 more
doaj   +1 more source

The Correlation between Junk Food Consumption and Age of Menarche of Elementary School Student In Gedung Johor Medan

open access: yesUnnes Journal of Public Health, 2018
Acceleration age of menarche, can increase the chances of endometrial hyperplasia. The incidence of uterine and breast cancer are associated with early menarche. Junk food is indicated as one of the triggers of early menarche. With observational-analytic
Surya Anita, Yunida Turisna Simanjuntak
doaj   +1 more source

The Relationship of Perceptions, Self-efficacy, and Junk Food Consumption With Demographic Variables in Female Students in Shahrekord [PDF]

open access: yesInternational Journal of Epidemiologic Research, 2018
Background and aims: Inappropriate or imbalanced consumption of foods may lead to several health problems, especially among adolescents, the complications of which will remain for several years. This study aimed to investigate perceptions, self-efficacy,
Elahe Tavassoli   +3 more
doaj   +1 more source

Tumour–host interactions in Drosophila: mechanisms in the tumour micro‐ and macroenvironment

open access: yesMolecular Oncology, EarlyView.
This review examines how tumour–host crosstalk takes place at multiple levels of biological organisation, from local cell competition and immune crosstalk to organism‐wide metabolic and physiological collapse. Here, we integrate findings from Drosophila melanogaster studies that reveal conserved mechanisms through which tumours hijack host systems to ...
José Teles‐Reis, Tor Erik Rusten
wiley   +1 more source

Epigenetic silencing of the liver‐specific lncRNA LUNAR promotes liver cancer progression via NOTCH activation

open access: yesMolecular Oncology, EarlyView.
LUNAR is a liver‐specific long noncoding RNA (lncRNA) that is highly expressed in normal liver but becomes epigenetically silenced in hepatocellular carcinoma through promoter hypermethylation. Loss of LUNAR is associated with NOTCH activation, epithelial–mesenchymal transition, and metastasis, whereas restoring LUNAR restrains metastatic progression ...
Se Ha Jang   +9 more
wiley   +1 more source

Junk Food [PDF]

open access: yes, 2015
Junk Food is a crime novel set in the wilds of suburban ...
Holt, Andrew
core   +1 more source

Bridging the gap: a genetically validated avian chorioallantoic membrane platform for investigation of spontaneous circulating tumor cells

open access: yesMolecular Oncology, EarlyView.
We established the avian chorioallantoic membrane (CAM) assay as a scalable in vivo model for studying circulating tumor cells (CTCs). Human gastrointestinal tumors spontaneously released genetically validated CTCs that were detected across multiple platforms, demonstrating that the CAM model provides an accessible tool for investigating early cancer ...
Dennis Roth   +17 more
wiley   +1 more source

The Birchwood Junk Food Cafe, Skelmersdale [PDF]

open access: yes, 2020
The University of Manchester conducted an evaluation of the Birchwood Junk Food Cafe, Skelmersdale, to assess the additional benefits of their adapted approach.
Steph Mitchell   +21 more
core   +1 more source

Loss of AMBRA1 activates MAPK and angiogenesis signaling pathways in melanoma cells

open access: yesFEBS Open Bio, EarlyView.
Loss of AMBRA1 in melanoma cells activates multiple oncogenic pathways associated with tumor progression. Transcriptomic and protein network analyses revealed that AMBRA1 depletion enhances MAPK/ERK signaling, angiogenesis, TGF‐β/EMT signaling, and Wnt/axon guidance pathways.
Milad Ibrahim   +4 more
wiley   +1 more source

Treatment with KCL‐286, a first‐in‐class retinoic acid receptor‐β (RARβ) agonist, ameliorates neuronal DNA damage and inflammation in a mouse model of Alzheimer's disease

open access: yesFEBS Open Bio, EarlyView.
Repair of neuronal DNA damage in Alzheimer's disease by KCL‐286. (A) Amyloid‐β oligomers and plaques impair neuronal DNA repair pathways, leading to DNA double‐strand breaks and glial activation. (B) KCL‐286 activates RARβ/RXR signalling via retinoic acid response elements (RAREs), associated with increased BRCA1 expression, enhanced DNA repair and ...
Natasha Hill   +6 more
wiley   +1 more source

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