Results 131 to 140 of about 5,024,889 (297)

An Evaluation of the Possible Protective Effects of Neonatal Striatal TransplantsAgainst Kainic Acid‐Induced Lesions [PDF]

, 1991
David R. Nash, Stanley M. Kaplan, Andrew B. Norman, Paul R. Sanberg   +3 more
openalex   +1 more source

Fetal Brain Transplantation in Kainic Acid Lesioned Caudate Nucleus of Adult Rats [PDF]

, 1992
Poonam Tandon, Velayudhan Mohan Kumar, Suneeta Verma, G. Gopinath, Ashok K. Shetty   +4 more
openalex   +1 more source

Do Fits Really Beget Fits? The Effect of Previous Epileptic Activity on the Subsequent Induction of the Tetanus Toxin Model of Limbic Epilepsy in the Rat

Neurobiology of Disease, 2001
The effect of pretreatment with either tetanus toxin (in ventral hippocampus) or kainic acid (into dorsal hippocampus, with or without suppression of seizures by phenobarbital) on the subsequent development of epilepsy in rats injected with tetanus toxin
J. Mellanby, A.J. Milward
doaj  

Inhibition by the adenosine analogue, (R‐)‐N⁶‐phenylisopropyl‐adenosine, of kainic acid neurotoxicity in rat hippocampus after systemic administration [PDF]

, 1993
D.G. MacGregor, Trevor W. Stone
openalex   +1 more source

Regulation of MAP2, GFAP, and calcium in the CA3 Region Following Kainic Acid Exposure to  organotypic hippocampal slice culture [version 3; peer review: 2 approved]

F1000Research
Background Neurodegeneration due to neurotoxicity is one of the phenomena in temporal lobe epilepsy. Experimentally, hippocampal excitotoxicity process can occur due to kainic acid exposure, especially in the CA3 area.
Aulanni'am Aulanni'am, Kusworini Handono, Ettie Rukmigarsari, Hidayat Sujuti, Machlusil Husna   +4 more
doaj  

Isolation, localization, and cloning of a kainic acid binding protein from frog brain. [PDF]

, 1990
Robert J. Wenthold, David R. Hampson, Keiji Wada, Chyren Hunter, Michael D. Oberdorfer, Claude J. Dechesne   +5 more
openalex   +1 more source

A short review on toxin-induced animal models of Huntington’s disease

Chronic Diseases Journal
BACKGROUND: Huntington's disease (HD) is a hereditary neurodegenerative motor and cognitive disorder. Different animal models of HD have been provided to study the different aspects of the disease, such as pathophysiology and treatment, and also the cure.
Ahmad Fotoohi
doaj   +1 more source

Histological Correlates of Auditory Nerve Injury from Kainic Acid in the Budgerigar (Melopsittacus undulatus). [PDF]

J Assoc Res Otolaryngol, 2023
Wang Y, Abrams KS, Youngman M, Henry KS.
europepmc   +1 more source

Corticotropin-releasing hormone (CRH) downregulates the function of its receptor (CRF1) and induces CRF1 expression in hippocampal and cortical regions of the immature rat brain. [PDF]

, 2002
In addition to regulating the neuroendocrine stress response, corticotropin-releasing hormone (CRH) has been implicated in both normal and pathological behavioral and cognitive responses to stress. CRH-expressing cells and their target neurons possessing
Baram, Tallie Z, Brunson, Kristen L, Grigoriadis, Dimitri E, Lorang, Marge T   +3 more
core  

Studies on the Ozone Oxidation Products of Kainic Acid and Its Isomers. (i) Isomerization of Methyl Ketone Compound. (1)

, 1956
Ritsuo Nakamori
openalex   +2 more sources