Results 181 to 190 of about 17,773 (204)
Some of the next articles are maybe not open access.
2007
Human infection by KSHV is associated with the development of at least three proliferative disorders: Kaposi’s sarcoma (KS), primary effusion lymphoma (PEL) and a subset of multicentric Castleman’s disease (MCD). In keeping with the classification of KSHV as a lymphotropic (γ2) herpesvirus, two of these (PEL and MCD) are primary disorders of the B cell
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Human infection by KSHV is associated with the development of at least three proliferative disorders: Kaposi’s sarcoma (KS), primary effusion lymphoma (PEL) and a subset of multicentric Castleman’s disease (MCD). In keeping with the classification of KSHV as a lymphotropic (γ2) herpesvirus, two of these (PEL and MCD) are primary disorders of the B cell
openaire +1 more source
Regulation of KSHV Lytic Gene Expression
2006The life cycle of KSHV, latency versus lytic replication, is mainly determined at the transcriptional regulation level. A viral immediate-early gene product, replication and transcription activator (RTA), has been identified as the molecular switch for initiation of the lytic gene expression program from latency.
H, Deng, Y, Liang, R, Sun
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Kaposi Sarcoma-Associated Herpesvirus (KSHV)
Trends in Microbiology, 2020Blossom Damania, Philip Lange
exaly
Kaposi sarcoma-associated herpesvirus (KSHV): Molecular biology and oncogenesis
Cancer Letters, 2010Blossom Damania
exaly
Update on KSHV epidemiology, Kaposi Sarcoma pathogenesis, and treatment of Kaposi Sarcoma
Cancer Letters, 2011Thomas Uldrick
exaly
KSHV/HHV8‐associated lymphomas
British Journal of Haematology, 2008Annunziata Gloghini, Antonino Carbone
exaly