Results 11 to 20 of about 4,323 (166)

Lambert-Eaton myasthenic syndrome: the 60th anniversary of Eaton and Lambert's pioneering article

open access: yesArquivos de Neuro-Psiquiatria, 2018
This historical review describes the contribution of Drs. Lee M. Eaton and Edward H. Lambert to the diagnosis of myasthenic syndrome on the 60th anniversary of their pioneering article (JAMA 1957) on the disease.
Paulo José Lorenzoni   +3 more
doaj   +3 more sources

Lung adenocarcinoma with Lambert–Eaton myasthenic syndrome indicated by voltage-gated calcium channel: a case report [PDF]

open access: yesJournal of Medical Case Reports, 2012
Introduction Lambert–Eaton myasthenic syndrome is a rare disorder and it is known as a paraneoplastic neurological syndrome. Small cell lung cancer often accompanies this syndrome.
Arai Hiromasa   +10 more
doaj   +2 more sources

Lambert-Eaton myasthenic syndrome: report of two cases [PDF]

open access: yesArquivos de Neuro-Psiquiatria, 1998
Two cases of Lambert-Eaton myasthenic syndrome, in female patients whose neoplasm investigation was negative, are reported. Repetitive stimulation of ulnar nerve showed an incremental response (+187% and +198%).
ROSANA H. SCOLA   +8 more
doaj   +2 more sources

Lambert-Eaton Syndrome in Children

open access: yesPediatric Neurology Briefs, 2014
Investigators from Boston Children's Hospital; the Lahey Clinic, Burlington, MA; and Ohio State University, report 3 children presenting between ages 9 and 10 years with proximal lower extremity weakness with areflexia and low-amplitude compound muscle ...
J Gordon Millichap, John J Millichap
doaj   +2 more sources

Adenocarcinoma of Lung Presenting as Lambert-Eaton Myasthenic Syndrome

open access: yesJournal of Investigative Medicine High Impact Case Reports, 2017
Lambert-Eaton myasthenic syndrome (LEMS) is a paraneoplastic neuromuscular junction disorder. LEMS presents with muscular weakness and fatigability, mainly involving the proximal lower limbs.
Sumera Bukhari MD   +4 more
doaj   +2 more sources

The Case of a 28‐Year‐Old Man With Gradually Progressive Proximal Leg Weakness

open access: yesAnnals of Clinical and Translational Neurology
This is a case of a 28‐year‐old man who presented with a 6‐month history of gradually progressive proximal leg weakness and pain that worsened on exercise and was relieved by rest. He had no symptoms in his upper limbs.
Melody T. Asukile   +3 more
doaj   +2 more sources

The association between Lambert–Eaton myasthenic syndrome and small cell lung carcinoma

open access: yesImmunoTargets and Therapy, 2013
Sarah EW Briggs,1 Paul Gozzard,2 Denis C Talbot31Department of Oncology, Oxford University Hospitals Trust, Churchill Hospital, Oxford, UK; 2Nuffield Department of Clinical Neurosciences, University of Oxford, John Radcliffe Hospital, Oxford UK ...
Briggs SEW, Gozzard P, Talbot DC
doaj   +1 more source

Electrophysiological evaluation of the neuromuscular junction: a brief review

open access: yesArquivos de Neuro-Psiquiatria, 2023
The nerve terminal and muscle membrane compose the neuromuscular junction. After opening the voltage-gated calcium channels, action potentials from the motor axons provoke a cascade for the acetylcholine release from synaptic vesicles to the synaptic ...
João Aris Kouyoumdjian   +1 more
doaj   +1 more source

Lambert-Eaton myasthenic syndrome against the background of thyroid cancer: a clinical case [PDF]

open access: yesКлиническая практика
BACKGROUND: Lambert–Eaton myasthenic syndrome (G73.1 according to ICD-10) is a rare autoimmune disease associated with a presynaptic impairment of neuromuscular transmission due to the production of antibodies to voltage-gated calcium channels.
Elena V. Khozhenko   +5 more
doaj   +1 more source

Lambert–Eaton myasthenic syndrome: Clinical review [PDF]

open access: yes, 2016
Lambert–Eaton myasthenic syndrome (LEMS) is an autoimmune disease of the neuromuscular junction and approximately 60% of LEMS patients have a tumor, mostly small cell lung cancer, as a paraneoplastic neurological syndrome.
Motomura, Masakatsu   +2 more
core   +1 more source

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