Results 201 to 210 of about 10,558,900 (341)

Cell(s) of Origin of Langerhans Cell Histiocytosis.

open access: yesHematology/Oncology Clinics of North America, 2015
M. Collin   +3 more
semanticscholar   +1 more source

Epidermal Keratinocyte Cells in Laboratory, Clinical Trials, and Market: A Narrative Review

open access: yesHealth Science Reports, Volume 8, Issue 10, October 2025.
ABSTRACT Background and Aims The skin serves essential protective and regulatory functions. In severe injuries such as burns, keratinocytes—being the main epidermal cells—play a pivotal role in skin regeneration through secretion of growth factors and cytokines.
Sona Zare   +4 more
wiley   +1 more source

Normal numbers of T6positive epidermal Langerhans cells across the leprosy spectrum

open access: bronze, 1984
R.B. Narayanan   +3 more
openalex   +1 more source

High prevalence of somatic MAP2K1 mutations in BRAF V600E-negative Langerhans cell histiocytosis.

open access: yesBlood, 2014
N. Brown   +6 more
semanticscholar   +1 more source

Pancreatic Damage in Ovarian Cancer–Associated Cachexia Is Driven by Activin A Signalling

open access: yesJournal of Cachexia, Sarcopenia and Muscle, Volume 16, Issue 5, October 2025.
ABSTRACT Background Cancer‐associated cachexia (CAC) is a severe metabolic disorder characterized by involuntary weight loss, skeletal muscle atrophy and adipose tissue depletion. It is a major contributor to morbidity and mortality in the advanced stages of various cancers.
Amirhossein Abazarikia   +5 more
wiley   +1 more source

Patients with langerhans cell histiocytosis and hypothalamic-pituitary involvement: insights from the HEROS study cohort. [PDF]

open access: yesPituitary
Masri Iraqi H   +11 more
europepmc   +1 more source

Somatic activating ARAF mutations in Langerhans cell histiocytosis.

open access: yesBlood, 2014
David S. Nelson   +14 more
semanticscholar   +1 more source

Diabetic Wound Repair: From Mechanism to Therapeutic Opportunities

open access: yesMedComm, Volume 6, Issue 10, October 2025.
Diabetic wound healing is compromised by chronic metabolic dysfunction that disrupts essential cellular processes. This review examines key predisposing factors, including neuropathy and vascular disease, characterizes dysfunction in critical cell types such as macrophages and fibroblasts, evaluates limitations of current models, compares established ...
Renyuan Wang   +5 more
wiley   +1 more source

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