Langerhans Cell Histiocytosis [PDF]
Sandra, Camelo-Piragua +2 more
openaire +2 more sources
Autoantigen mRNA‐LNP Vaccination Drives Therapeutic Efficacy in Preclinical Models for Autoimmunity
Systemic and intramuscular delivery of autoantigen mRNA via lipid nanoparticles reprograms antigen‐presenting cells toward a mature, homeostatic state, driving antigen‐specific T cell exhaustion and providing therapeutic efficacy across autoimmune disease models.
Paulien Baeten +30 more
wiley +1 more source
ABSTRACT Introduction Children treated with ifosfamide seem to have an increased risk for developing chronic kidney disease (CKD) already within the first years following chemotherapy. However, studies investigating CKD prevalence in this specific phase are scarce.
Lotte Pitlo +9 more
wiley +1 more source
Distinct postnatal trajectories of mouse dendritic epidermal T cells and Langerhans cells independent of microbiota. [PDF]
Obwegs D +23 more
europepmc +1 more source
Effect of ultrasound treatment of the skin on activation of Langerhans cells and antibody production in rodents. [PDF]
Enjo S +4 more
europepmc +1 more source
Mechanistic Pathways of Wound Healing: From Hemostasis to Scar‐Free Regeneration
ABSTRACT Wound healing is the critical process regulated through a harmonious coordination of cellular, molecular, pathological, and systemic communications. Normally, healing occurs in four overlapping phases, which are modified and controlled by various growth factor interactions and signaling pathways.
Devadass Jessy Mercy +2 more
wiley +1 more source
Antibody-mediated targeting of SOSIP HIV-1 Env to skin Langerhans cells potentiates humoral responses. [PDF]
Hammoudi A +23 more
europepmc +1 more source
Cross-presenting Langerhans cells are required for the early reactivation of resident CD8+ memory T cells in the epidermis. [PDF]
Kamenjarin N +13 more
europepmc +1 more source
IL‐22Rα1 restrains pancreatic injury independently of its C‐terminal STAT3‐amplifying domain
IL‐22R, and to a lesser extent IL‐22, are key drivers of protection in acute pancreatitis. Although the C‐terminal domain of IL‐22R enhances STAT3 activation in the pancreas, it is not required for disease protection. Classical IL‐22R signaling alone is sufficient to control inflammation and promote pancreatic recovery.
Léna Puigdevall +4 more
wiley +1 more source

