Results 121 to 130 of about 14,552 (222)
Inhibition of HSP90 distinctively modulates the global phosphoproteome of <i>Leishmania mexicana</i> developmental stages. [PDF]
Porta EO +4 more
europepmc +1 more source
Leishmania mexicana centrin knockout parasites promote M1-polarizing metabolic changes. [PDF]
Volpedo G +9 more
europepmc +1 more source
Complex cutaneous infection by Leishmania mexicana treated with miltefosine. [PDF]
Sadowsky RL, Emerson CM, Reid DC.
europepmc +1 more source
Localized cutaneous leishmaniasis (LCL), known as "chiclero's ulcer" in southeast Mexico, was described by Seidelin in 1912. Since then, the sylvatic region of the Yucatan peninsula has been identified as an endemic focus of LCL.
Andrade-Narváez Fernando J +3 more
doaj
CXCR5 and TIM-3 expressions define distinct exhausted T cell subsets in experimental cutaneous infection with Leishmania mexicana. [PDF]
Diupotex M +4 more
europepmc +1 more source
In the Yucatan Peninsula, Mexico, localized cutaneous leishmaniasis (LCL) caused by Leishmania (Leishmania) mexicana is a typical wild zoonosis restricted to the forest, and humans are only accidentally involved. The transmission of L.
Andrade-Narvaez Fernando J +5 more
doaj
Catalase impairs Leishmania mexicana development and virulence. [PDF]
Sádlová J +15 more
europepmc +1 more source
Three types of Leishmania mexicana amastigotes: Proteome comparison by quantitative proteomic analysis. [PDF]
Pacakova L +3 more
europepmc +1 more source
Immunity to Leishmania mexicana parasite [PDF]
Background: Cutaneous Leishmaniasis caused by the Leishmania mexicana complex is associated with unpleasant or disfiguring lesions, for which there only limited treatment options. The life cycle of L. mexicana consists of two stages which involve different immune evasion mechanisms: promastigote and amastigote. Understanding parasitic interactions with
openaire

