Results 241 to 250 of about 27,333,880 (308)

IFNγ-secreting T cells that highly express IL-2 potently inhibit the growth of intracellular M. tuberculosis in macrophages. [PDF]

Front Immunol
Zhu L, Wang B, Gu J, Zhou J, Wu Y, Xu W, Yang M, Cai X, Shen H, Lu L, Wang F.   +10 more
europepmc   +1 more source

LncRNA SNHG16 Inhibits Intracellular M. tuberculosis Growth Involving Cathelicidin Pathway, Autophagy, and Effector Cytokines Production. [PDF]

ACS Omega
Huang G, Wu X, Ji X, Peng Y, Wang J, Cai X, Wang Y, Yang E, Zhu L, Wu Y, Sun Q, Shen L, Sha W, Shen H, Wang F.   +14 more
europepmc   +1 more source

M. tuberculosis meets European Lead Factory – identification and structural characterization of novel Rv0183 inhibitors using X-ray crystallography

Riegler-Berket L, Gödl L, Polidori N, Aschauer P, Grininger C, Prosser G, Lichtenegger J, Sagmeister T, Parigger L, Gruber C, Reiling N, Oberer M.   +11 more
europepmc   +1 more source

Autologous human iPSC-derived Alveolus-on-Chip reveals early pathological events of M. tuberculosis infection

Luk CH, Conway GL, Goh KJ, Fearns A, Rodriguez-Hernandez I, Day NJ, Athanasiadi N, D’Antuono R, Pellegrino E, Stucki JD, Hobi N, Gutierrez MG.   +11 more
europepmc   +1 more source

Subpopulations in clinical samples of M. tuberculosis can give rise to rifampicin resistance and shed light on how resistance is acquired

Brunner VM, Fowler PW.
europepmc   +1 more source

M. tuberculosis invades and disrupts the blood brain barrier directly to initiate meningitis

Proust A, Wilkinson KA, Wilkinson RJ.
europepmc   +1 more source

An improved catalogue for whole-genome sequencing prediction of bedaquiline resistance in M. tuberculosis using a reproducible algorithmic approach

Adlard D, Joseph L, Webster H, O’Reilly A, Knaggs J, Peto TE, Crook DW, Omar SV, Fowler PW.   +8 more
europepmc   +1 more source

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Innate immune responses to M. tuberculosis infection

Tuberculosis, 2011
A prerequisite for successful establishment of Mycobacterium tuberculosis in the host is its ability to survive after internalization in alveolar macrophages that they encounter after inhalation. The innate immune response protects some individuals to the extent that they remain uninfected.
Krishnamurthy, Natarajan, Manikuntala, Kundu, Pawan, Sharma, Joyoti, Basu   +3 more
openaire   +3 more sources

M. tuberculosis persistence, latency, and drug tolerance

Tuberculosis, 2004
The success of Mycobacterium tuberculosis as a pathogen is largely attributable to its ability to persist in host tissues, where drugs that are rapidly bactericidal in vitro require prolonged administration to achieve comparable effects. Latency is a frequent outcome of untreated or incompletely treated M.
James E, Gomez, John D, McKinney
openaire   +3 more sources