Results 111 to 120 of about 17,128 (260)
Response to: "Considerations on the Histologic Definition and Clinical Translation of Burnt-Out MASLD in Resected HCC". [PDF]
Watanabe Y +3 more
europepmc +1 more source
Summary of a multicentre TriNetX study evaluating outcomes after percutaneous coronary intervention (PCI) in patients with cirrhosis. Cirrhosis was associated with higher 1‐year gastrointestinal bleeding and mortality, especially with decompensated disease.
Dhir Gala +13 more
wiley +1 more source
Correspondence on Editorial regarding “Identification of signature gene set as highly accurate determination of MASLD progression” [PDF]
Sungju Jung +4 more
doaj +1 more source
Does Burnt-Out MASLD Mark Loss of Hepatic Lipid-Buffering Capacity? [PDF]
Toshikuni N.
europepmc +1 more source
Metabolic dysfunction‐associated steatohepatitis is the second commonest US indication for liver transplantation but remains largely undiagnosed and untreated. Fibrosis stage is the strongest predictor of major adverse liver outcomes, with decompensation rates and transplant‐free survival diverging sharply between F3 and F4, while cardiovascular ...
Malek Ayoub +2 more
wiley +1 more source
Inhibition of Acetylation Activity of p300/CBP Ameliorates Hepatic Steatosis Through Downregulation of ACSL4. [PDF]
Cai LY +7 more
europepmc +1 more source
Impact of incretin analogues on lipid and lipoprotein metabolism in obesity and diabetes
Abstract Incretin‐based therapies have gained momentum as a key strategy for reducing cardiovascular risk in individuals with obesity and/or type 2 diabetes (T2D). It remains unclear whether the cardiovascular benefits reflect a direct reduction in atherogenic lipoproteins—namely, low‐density lipoproteins (LDL), very low‐density lipoproteins (VLDL) and
Andrea Baragetti, Giuseppe Danilo Norata
wiley +1 more source
Letter to the Editor: When ALT defines the study denominator-MASLD burden or metabolic liver-injury burden? [PDF]
Cheng X, Sun M.
europepmc +1 more source
Obesity is a chronic, relapsing, multisystem disease in which cardiometabolic risk arises from excess adiposity and progressive dysfunction of peripheral organs, ultimately disrupting endocrine and metabolic crosstalk among tissues. Within this network, sulphur‐based biology, centred on hydrogen sulphide and related reactive sulphur species, has ...
Martina Smimmo +4 more
wiley +1 more source

